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  • 11
    Digitale Medien
    Digitale Medien
    Influence of micromixing on the free-radical polymerization in a discontinuous process (1994)
    Weinheim : Wiley-Blackwell
    Macromolecular Theory and Simulations 3 (1994), S. 855-883 
    Details
    ISSN: 1022-1344
    Schlagwort(e): Chemistry ; Polymer and Materials Science
    Quelle: Wiley InterScience Backfile Collection 1832-2000
    Thema: Chemie und Pharmazie , Physik
    Notizen: With the help of a simple reaction-diffusion model with constant striation thickness the influence of micromixing on free-radical polymerization was investigated for several test reactions with discontinuous prepolymerization and jerky addition of selected reactants. Monomer conversion or mean values of molar mass and chemical composition cannot be expected to be very sensitive to micromixing effects. If molar mass distributions are to be used, problems will arise from the fact that the distribution of the polymer accumulated during prepolymerization covers mixing influences occurring after reactant feed. The instantaneous molar mass distribution would be more suitable. Time-integral distributions of chemical composition or sequence length in combination with appropriate test reactions proved to be feasible indicators for the effects of micromixing as it becomes possible to separate the distribution of the prepolymer from that of the polymer which is formed after addition when micromixing is to be investigated.
    Zusätzliches Material: 31 Ill.
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1002/mats.1994.040030504
    Standort Signatur Einschränkungen Verfügbarkeit
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    Artikel (Nationallizenzen)
    Volltext
  • 12
    Digitale Medien
    Digitale Medien
    kunstformen der natur (1992)
    Weinheim : Wiley-Blackwell
    Biologie in unserer Zeit 22 (1992), S. 317-317 
    Details
    ISSN: 0045-205X
    Schlagwort(e): Life and Medical Sciences
    Quelle: Wiley InterScience Backfile Collection 1832-2000
    Thema: Biologie
    Materialart: Digitale Medien
    URL: http://dx.doi.org/10.1002/biuz.19920220606
    Standort Signatur Einschränkungen Verfügbarkeit
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    Artikel (Nationallizenzen)
    Volltext
  • 13
    facet.materialart.
    Unbekannt
    Untersuchungen zur Identifizierung und Wirkung des Sexualpheromons dekapoder Krebse unter besonderer Berücksichtigung der Strandkrabbe Carcinus maenas (1980)
    In:  (PhD/ Doctoral thesis), Christian-Albrechts-Universität Kiel, Kiel, Germany, 100 pp
    Details
    Publikationsdatum: 2018-01-17
    Materialart: Thesis , NonPeerReviewed
    Format: text
    Standort Signatur Einschränkungen Verfügbarkeit
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    OceanRep: Hochschulschrift - nicht über Verlag veröffentlicht
  • 14
    facet.materialart.
    Unbekannt
    Expanded spectrum of exon 33 and 34 mutations in SRCAP and follow-up in patients with Floating-Harbor syndrome (2014)
    BioMed Central
    Details
    Publikationsdatum: 2014-12-06
    Beschreibung: Background: Floating-Harbor syndrome is a rare autosomal dominant short stature syndrome with retarded speech development, intellectual disability and dysmorphic facial features. Recently dominant mutations almost exclusively located in exon 34 of the Snf2-related CREBBP activator protein gene were identified to cause FHS. Methods: Here we report the genetic analysis of 5 patients fulfilling the diagnostic criteria of FHS obtained by Sanger sequencing. All of them presented with short stature, speech delay as well as psychomotor delay and typical facial dysmorphism. Three patients showed a good response to growth hormone treatment. Results: Two patients demonstrate novel, heterozygous de novo frameshift mutations in exon 34 (c.7396delA and c.7218dupT) leading to premature stop mutations in SRCAP (p.Val2466Tyrfs*9 and p.Gln2407Serfs*36, respectively). In two further patients we found already known SRCAP mutations in exon 34, c.7330C 〉 T and c.7303C 〉 T, respectively, which also lead to premature stop codons: p.Arg2444* and p.Arg2435*. In one patient, we identified a novel de novo stop mutation in exon 33 (c.6985C 〉 T, p.Arg2329*) demonstrating that not all FHS cases are caused by mutations in exon 34 of SRCAP. Conclusions: Our data confirm a mutational hot spot in the final exon of SRCAP in the majority of FHS patients but also show that exon 33 of this gene can be affected.
    Digitale ISSN: 1471-2350
    Thema: Biologie , Medizin
    Publiziert von BioMed Central
    Standort Signatur Einschränkungen Verfügbarkeit
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