In:
Molecular Oncology, Wiley, Vol. 10, No. 6 ( 2016-06), p. 775-788
Abstract:
GPER is negatively correlated with the progression of TNBC. G‐1 inhibits the in vitro motility of TNBC cells via suppression of EMT. The inhibition of NF‐κB mediates G‐1 induced EMT suppression. GSK‐3β is involved in G‐1 suppressed NF‐κB activities. PI3K/Akt and ERK1/2 are involved in G‐1 induced phosphorylation of GSK‐3β.
Type of Medium:
Online Resource
ISSN:
1574-7891
,
1878-0261
DOI:
10.1016/j.molonc.2016.01.002
Language:
English
Publisher:
Wiley
Publication Date:
2016
detail.hit.zdb_id:
2322586-5