In:
Developmental Dynamics, Wiley, Vol. 246, No. 10 ( 2017-10), p. 759-769
Abstract:
Loss of zebrafish tbx6l alone has little to no effect on embryonic development because tbx16, a closely related gene, compensates for loss of tbx6l function. Loss of tbx6l enhances the tbx16 mutant phenotype, revealing that tbx6l functions redundantly with tbx16 during tail paraxial mesoderm development. Double mutant analyses reveal that tbx6l does not appear to genetically interact with ta or tbx6, two other mesodermally‐expressed T‐box genes. In addition, tbx6l loss does not enhance the supernumerary somite phenotype of mesogenin1 mutants.
Type of Medium:
Online Resource
ISSN:
1058-8388
,
1097-0177
DOI:
10.1002/dvdy.v246.10
Language:
English
Publisher:
Wiley
Publication Date:
2017
detail.hit.zdb_id:
1473797-8
SSG:
12