Interleukin-13 Inhibits Lipopolysaccharide-Induced BPIFA1 Expression in Nasal Epithelial Cells
Fig 2
Phosphorylated p65 does not respond to LPS-mediated BPIFA1 expression.
(A) RPMI-2650 cells were treated with LPS (10 μg/ml) for the indicated times, and the expression levels of phosphorylated ERK, JNK, p38, and p65 were determined by western blot. β-actin was used as the loading control. (B) Protein expression levels were quantified with densitometric analysis and presented as mean ± standard deviation, derived from three independent experiments.