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1

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Seismic geomorphology : applications to hydrocarbon exploration and production (2007)

Davies, R. J. ; Posamentier, Henry W. 1948- ; Wood, L.J. ; [et al.]

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Keywords: Seismic prospecting ; Petroleum Prospecting ; Geomorphology ; Hydrocarbons ; Three-dimensional imaging ; Konferenzschrift ; Aufsatzsammlung ; Prospektion ; Geomorphologie ; Kohlenwasserstofflagerstätte ; Dreidimensionale Seismik ; Seismik ; Seismische Prospektion ; Seismische Stratigraphie

Type of Medium: Online Resource

Pages: 1 Online-Ressource (274 Seiten)

ISBN: 1862392234 , 9781862392236

Series Statement: Geological Society special publication 277

URL: http://sp.lyellcollection.org/content/277/1

DDC: 551.41

RVK:

RB 10256

RVK:

UT 1200

Language: English

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2

Electronic Resource

An improved method of adjusting flour moisture in studies on lipid binding (1969)

DAVIES, R. J. ; DANIELS, N. W. R. ; GREENSHIELDS, R. N.

Oxford, UK : Blackwell Publishing Ltd

International journal of food science & technology 4 (1969), S. 0

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ISSN: 1365-2621

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Process Engineering, Biotechnology, Nutrition Technology

Notes: Summary. A method is described for the controlled adjustment of flour moisture level over the range 14-60%. Water was added as powdered ice to flour dispersed in liquid nitrogen thereby avoiding the introduction of mechanical work. the effect of moisture on the distribution of free and bound lipids in a hard wheat flour was investigated using this technique. Free lipid decreased above 20% moisture, while extractable bound lipid increased only above 25% moisture. These different critical moisture levels are considered with reference to the distribution of neutral and polar lipid fractions in the moistened flour.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1365-2621.1969.tb01504.x

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3

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A method for reconstituting fat extracted flour (1970)

DAVIES, R. J. ; DANIELS, N. W. R. ; GREENSHIELDS, R. N.

Oxford, UK : Blackwell Publishing Ltd

International journal of food science & technology 5 (1970), S. 0

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ISSN: 1365-2621

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Process Engineering, Biotechnology, Nutrition Technology

Notes: Summary. A low temperature technique is described for the reconstitution of fat extracted wheat flour which allows the return of lipid material with the minimum alteration of flour properties. A study of the effect of moisture level on lipid binding in reconstituted flour is compared with a similar study of the original flour. Flours with augmented lipid contents were produced and confirmation that the method avoided the introduction of mechanical work obtained.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1365-2621.1970.tb01552.x

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4

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A reply (1976)

Lack, J. A. ; Davies, R. J.

Oxford, UK : Blackwell Publishing Ltd

Anaesthesia 31 (1976), S. 0

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ISSN: 1365-2044

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1365-2044.1976.tb11912.x

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5

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Guidelines for the standardization of bronchial provocation tests with allergens (1991)

Melillo, G. ; Aas, K. ; Cartier, A. ; [et al.]

Oxford, UK : Blackwell Publishing Ltd

Allergy 46 (1991), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1398-9995.1991.tb00594.x

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6

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Inhaled and nasal corticosteroids: safety aspects* (2000)

Passalacqua, G. ; Albano, M. ; Canonica, G. W. ; [et al.]

Copenhagen : Munksgaard International Publishers

Allergy 55 (2000), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1034/j.1398-9995.2000.00370.x

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7

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Allergen/irritant interaction - its role in sensitization and allergic disease (1998)

Devalia, J. L. ; Rusznak, C. ; Davies, R. J.

Oxford, UK : Blackwell Publishing Ltd

Allergy 53 (1998), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1398-9995.1998.tb03903.x

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8

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Mechanisms of pollution-induced airway disease: in vitro itudies in the upper and lower airways (1997)

DEVALIA, J. L. ; BAYRAM, H. ; RUSZNAK, C. ; [et al.]

Oxford, UK : Blackwell Publishing Ltd

Allergy 52 (1997), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Notes: Devalia JL, Bayram H, Rusznak C, Calderón M, Sapsford RJ, Abdelaziz MA, Wang J, Davies RJ. Mechanisms of pollution-induced airway disease: in vitro studies in the upper and lower airways.Evidence from both epidemiological and laboratory-based studies suggests that increased exposure to liquid petroleum and gas-derived air pollutants [nitrogen dioxide (NO2), ozone, and respirable particulate matter] may play a role in the clinical manifestation of both allergic and non-allergic airway disease. The mechanisms and cell types involved in pollutant-mediated effects in the airways, however, are not clear. In vitro studies have suggested that human fibroblasts, B-lymphocytes, alveolar macrophages, and epithelial cells/cell lines may be involved. Studies of fibroblasts and macrophages have demonstrated that exposure to ozone results in decreased cell viability and increased release of pro-inflammatory mediators from macrophages. Similarly, studies of B-lymphocytes have demonstrated that exposure to diesel exhaust particles (DEP) enhances the synthesis of immunoglobulin E by these cells. The airway epithelial cells have received the greatest attention in mechanistic studies of air pollution-induced airway disease and suggest that these cells are likely to play a pivotal role in the pathogenesis of airways disease. Various studies have demonstrated that exposure of nasal or bronchial epithelial cells to NO2, ozone, and DEP results in significant synthesis and release of pro-inflammatory mediators, including eicosanoids, cytokines, and adhesion molecules. Additionally, evidence suggests that epithelial cells of atopic individuals release significantly greater amounts of cytokines such as granulocyte-macrophage colony stimulating factor (GM-CSF), interleukin-6 (IL-6), IL-8, and regulated on activation, normal T-cell expressed and secreted (RANTES), on exposure to NO2 and ozone. Studies investigating the biological relevance of epithelial cell-derived pro-inflammatory mediators have shown that these enhance eosinophil chemotaxis and eosinophil adherence to endothelial cells, suggesting that pollution-induced inflammation of the airways is likely to be influenced by modulation of epithelial synthesis and release of these mediators.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1398-9995.1997.tb04870.x

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9

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Allergen-irritant interaction and the role of corticosteroids (1997)

DAVIES, R. J. ; RUSZNAK, C. ; CALDERON, M. A. ; [et al.]

Oxford, UK : Blackwell Publishing Ltd

Allergy 52 (1997), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Notes: Davies RJ, Rusznak C, Calderón MA, Wang JH, Abdelaziz MM, Devalia JL. Allergen-irritant interaction and the role of corticosteroids.Studies of exposure to air pollutants, such as ozone and nitrogen dioxide (NO2)± sulphur dioxide (SO2), have demonstrated that these agents, either individually or in combination, increase the airway response of both asthmatics and allergic rhinitics to inhaled allergen. Other studies have demonstrated that exposure to these pollutants significantly increased the levels of eosinophil cationic protein (ECP) in the nasal secretions of both asthmatics and allergic rhinitics, suggesting that pollutants may prime eosinophils for subsequent activation by allergen. More recently, our studies have demonstrated that treatment with inhaled corticosteroids, such as fluticasone propionate, significantly attenuated pollution+ allergen-induced release of ECP in allergic rhinitics. Although the mechanisms underlying the potentiating effects of pollutants on allergen-induced changes in the airways of allergic individuals are not fully understood, in vitro studies have suggested that airway epithelial cells may play an important role, since they can synthesize a variety of cytokines and adhesion molecules which influence the activity of eosinophls and other inflammatory cells. Studies of nasal epithelial cells cultured from biopsies of atopic rhinitic and atopic non-rhinitic individuals have shown that they constitutively release significantly greater quantities of pro-inflammatory cytokines than nasal epithelial cells of non-atopic individuals, and that the release of these cytokines is greater from cells of atopic rhinitics during the pollen season. Furthermore, exposure of the cells of rhinitics to ozone led to an even greater release of these cytokines, and this effect was attenuated by treatment with fluticasone propionate and beclomethasone dipropionate.Taken together, the results described above provide evidence to suggest that atmospheric pollutants, such as ozone or the combination of NO2 and SO2, increase the bronchial response of mild asthmatic subjects to inhaled allergen, and that this may persist for a period of 24–48 h after exposure. Acute exposure to ozone or NO2 ‘primes’ the eosinophils for subsequent activation by allergen, and pretreatment with FP attenuates both the pollution-induced eosinophil priming and allergen-induced increase in the number of EGZstaining cells in the bronchial mucosa, suggesting that FP treatment may modulate airway inflammation by influencing the activity of eosinophils, and perhaps other inflammatory cell types.At the cellular level, airway epithelial cells probably play an important role in the initiation and maintenance of airway inflammation, since these cells express and release pro-inflammatory mediators which influence the recruitment and activation of inflammatory cells, such as neutrophils and eosinophils, in the airways. Nasal epithelial cells from both atopic non-rhinitics and atopic rhinitics release sigmficantly greater amounts of IL-8, GM-CSF and TNFα, than cells from non-atopic non-rhinitics. The cells of atopic rhinitics release significantly greater quantities of these cytokines during the pollen season, compared with outside of the pollen season, and exposure of these cells to ozone further enhances the release of these cytokines, an effect which can be attenuated by incubating the cells with FP or BDI?

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1398-9995.1997.tb04873.x

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10

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A double-blind study of hyposensitization with an alginate- conjugated extract of Dermatophagoides pteronyssinus (Conjuvac®) in patients with perennial rhinitis (1990)

Pastorello, E. A. ; Ortolani, C. ; Incorvaia, C. ; [et al.]

Oxford, UK : Blackwell Publishing Ltd

Allergy 45 (1990), S. 0

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ISSN: 1398-9995

Source: Blackwell Publishing Journal Backfiles 1879-2005

Topics: Medicine

Notes: In a 2-year double-blind placebo controlled study an immunological evaluation was carried out on 33 patients (15 males, 18 females, mean age 29.2 years) with mite-induced perennial rhinitis who were submitted to specific immunotherapy (IT) with an alginate-conjugated extract of D. pteronyssinus. The behaviour of IgE, IgG, IgG1, and IgG4, antibodies specific to D. pteronyssinus and its major allergen Der p1, was characterized by assessment of their changes m serum, and changes in IgG in nasal secretions during the treatment. The placebo-treated patients did not show any significant variation in the levels of specific antibodies, while in the actively treated patients we found; a statistically significant decrease (P 〈 0.005) of specific IgE, a statistically significant increase of specific IgG (P 〈 0.005), IgG1, (P 〈 0.005) and IgG4 (P 〈 0.005) in serum and a statistically significant increase (P 〈 0.001) of specific IgG in nasal secretions. The IgG response showed an early relative predominance of the IgG1 subclass and a late absolute predominance of IgG4 subclass, that confirmed the model of IgG4 restriction in prolonged allergen stimulation. No correlation was found between immunological and clinical data.

Type of Medium: Electronic Resource

URL: http://dx.doi.org/10.1111/j.1398-9995.1990.tb00526.x

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