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  • 1
    Online Resource
    Online Resource
    Bessel Processes, Schramm-Loewner Evolution, and the Dyson Model. (2016)
    Singapore :Springer Singapore Pte. Limited,
    Details
    Keywords: Statistical physics. ; Electronic books.
    Type of Medium: Online Resource
    Pages: 1 online resource (149 pages)
    Edition: 1st ed.
    ISBN: 9789811002755
    Series Statement: SpringerBriefs in Mathematical Physics Series ; v.11
    URL: https://ebookcentral.proquest.com/lib/geomar/detail.action?docID=4398759
    DDC: 530.15/29434
    Language: English
    Note: Intro -- Preface -- Contents -- 1 Bessel Processes -- 1.1 One-Dimensional Brownian Motion (BM) -- 1.2 Martingale Polynomials of BM -- 1.3 Drift Transformation -- 1.4 Quadratic Variation -- 1.5 Stochastic Integration -- 1.6 Itô's Formula -- 1.7 Complex Brownian Motion and Conformal Invariance -- 1.8 Stochastic Differential Equations for Bessel Processes -- 1.9 Kolmogorov Equation -- 1.10 BES(3) and Absorbing BM -- 1.11 BES(1) and Reflecting BM -- 1.12 Critical Dimension Dc=2 -- 1.13 Bessel Flow and Another Critical Dimension overlineDc=3/2 -- 1.14 Hypergeometric Functions Representing Bessel Flow -- References -- 2 Schramm--Loewner Evolution (SLE) -- 2.1 Complexification of Bessel Flow -- 2.2 Schwarz--Christoffel Formula and Loewner Chain -- 2.3 Three Phases of SLE -- 2.4 Cardy's Formula -- 2.5 SLE and Statistical Mechanics Models -- References -- 3 Dyson Model -- 3.1 Multivariate Extension of Bessel Process -- 3.2 Dyson Model as Eigenvalue Process -- 3.3 Dyson Model as Noncolliding Brownian Motion -- 3.4 Determinantal Martingale Representation (DMR) -- 3.5 Reducibility of DMR and Correlation Functions -- 3.5.1 Density Function ρξ(t, x) -- 3.5.2 Two-Time Correlation Function ρξ(s, x -- t, y) -- 3.6 Determinantal Process -- 3.7 Constant-Drift Transformation of Dyson Model -- 3.8 Generalization for Initial Configuration with Multiple Points -- 3.9 Wigner's Semicircle Law and Scaling Limits -- 3.9.1 Wigner's Semicircle Law -- 3.9.2 Bulk Scaling Limit and Homogeneous Infinite System -- 3.9.3 Soft-Edge Scaling Limit and Spatially Inhomogeneous Infinite System -- 3.10 Entire Functions and Infinite Particle Systems -- 3.10.1 Nonequilibrium Sine Process -- 3.10.2 Nonequilibrium Airy Process -- 3.11 Tracy--Widom Distribution -- 3.11.1 Distribution Function of the Maximum Position of Particles -- 3.11.2 Integrals Involving Resolvent of Correlation Kernel. , 3.11.3 Nonlinear Third-Order Differential Equation -- 3.11.4 Soft-Edge Scaling Limit -- 3.11.5 Painlevé II and Limit Theorem of Tracy and Widom -- 3.12 Beyond Determinantal Processes -- References -- Index.
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  • 2
    Online Resource
    Online Resource
    Elliptic Extensions in Statistical and Stochastic Systems. (2023)
    Singapore :Springer Singapore Pte. Limited,
    Details
    Keywords: Electronic books.
    Type of Medium: Online Resource
    Pages: 1 online resource (134 pages)
    Edition: 1st ed.
    ISBN: 9789811995279
    Series Statement: SpringerBriefs in Mathematical Physics Series ; v.47
    URL: https://ebookcentral.proquest.com/lib/geomar/detail.action?docID=7236610
    DDC: 515.983
    Language: English
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  • 3
    Electronic Resource
    Electronic Resource
    Inhibition Of Kinin Degradation On The Luminal Side Of Renal Tubules Reduces High Blood Pressure In Deoxycorticosterone Acetate Salt-Treated Rats (2000)
    Melbourne, Australia : Blackwell Science Pty
    Clinical and experimental pharmacology and physiology 27 (2000), S. 0 
    Details
    ISSN: 1440-1681
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: 1. To determine whether the antihypertensive response in deoxycorticosterone acetate (DOCA) salt-treated rats was mediated by kinins on the luminal side of renal tubules or in the circulation, selective urinary kininase inhibitors were administered to normal Brown Norway Kitasato (BN-Ki) rats and kininogen-deficient Brown Norway Katholiek (BN-Ka) rats.2. Kinins were degraded by neutral endopeptidase (NEP) and carboxypeptidase Y-like kininase (CPY) in urine, but were inactivated mainly by angiotensin-converting enzyme (ACE) in the plasma.3. Ebelactone B inhibited CPY, while poststatin inhibited CPY and NEP.4. Daily administration of poststatin (5 mg/kg per day, s.c.) for 3 days reduced blood pressure (BP) in DOCA salt-treated BN-Ki rats, but not in BN-Ka rats.5. Ebelactone B (5 mg/kg per day, s.c.) also reduced BP in BN-Ki rats, which was accompanied by increased urinary sodium excretion, but had no effect on BP in BN-Ka rats.6. Lisinopril (5 mg/kg per day, s.c.) had no effect on BP in either rat strain.7. Arterial kinin levels in BN-Ki rats increased significantly (2.2–4.6 pg/mL) with captopril (10 mg/kg, s.c.). However, arterial kinin levels that induced hypotension following the infusion of bradykinin (1000 ng/kg per min, i.v.) were 110- fold higher than endogenous arterial kinin levels attained following captopril.8. These results suggest that inhibition of kinin degradation on the luminal side of the renal tubules may effectively attenuate hypertension.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1046/j.1440-1681.2000.03209.x
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  • 4
    Electronic Resource
    Electronic Resource
    Clinical significance of bradykinin liberation during cardiopulmonary bypass and its prevention by a kallikrein inhibitor (1975)
    Springer
    Surgery today 5 (1975), S. 222-233 
    Details
    ISSN: 1436-2813
    Keywords: bradykinin ; cardiopulmonary bypass ; kininogen ; postperfusion lung syndrome ; kallikrein inhibitor ; Trasylol ; capillary permeability ; peripheral vasodilatation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Activation of the kinin system and effects of Trasylol (Bayer, A.G. Lebukusen, West Germany), a kallikrein inhibitor, were investigated on 52 patients during hemodilutional cardiopulmonary bypass (CPB). Immediately after the start of CPB, neither elevation of bradykinin nor reduction of plasma kininogen (KGN: a precursor of bradykinin) were observed. During CPB, bradykinin level in the blood was markedly elevated, correlating with the significant decrease of kininogen (p〈0.001). The longer the CPB time, the more marked the reduction of KGN. In the cases requiring over 60 minutes of CPB, the amounts of bradykinin released (4.6–18.0ng/ml) were sufficient to increase capillary permeability as well as peripheral vasodilatation. As shown by the significant increase of hematocrit (p〈0.005) and the extreme reduction of vascular resistance found at the end of CPB in the prolonged cases. Infusion of Trasylol into the extracorporeal circuit actually prevented the reduction of kininogen and the increase of hematocrit as well as the extreme decrease of vascular resistance in the cases of over 60 minutes CPB. These results clearly point out that Trasylol is beneficial for the prevention of bradykinin liberation and capillary permeability increase and for the maintenance of optimum peripheral vascular tone during CPB. Furthermore, the significance of these findings with regards to complications during and after prolonged CPB was discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02469765
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  • 5
    Electronic Resource
    Electronic Resource
    Direct observation of microcirculation of the basal region of rat gastric mucosa (1995)
    Springer
    Journal of gastroenterology 30 (1995), S. 557-564 
    Details
    ISSN: 1435-5922
    Keywords: intravital microscopy ; gastric mucosal microcirculation ; basal part of the gastric mucosa
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract We modified and improved techniques for the intravital microscopic observation of the rat gastric microcirculation. The stomach of anesthetized rats was cut along the greater curvature, and the posterior wall of the glandular stomach was fixed in a chamber with the serosal side up and perfused with warmed Tyrode's solution. A portion of the muscularis externa was resected with the serosa to make an observation window. Vascular casts were studied histologically after the injection of Monastral blue B gelatin solution. Vascular casts revealed that most of the microvasculature observed in the window was not located in the submucosa, but in the basal part of the mucosa. Microscopic observation showed that the basal mucosal arterioles branched to form the mucosal capillaries, and the collecting venules from the mucosal surface were seen in cross-sections to drain into the venules located in the basal mucosa, without penetrating the muscularis mucosae. Topical application of acetylcholine (0.03–10μM) to the window dilated the arterioles, and topical application of epinephrine (0.03–3μM) constricted them dose-dependently without affecting the collecting venules and the venules. This method made possible the direct observation of the microvasculature in the basal mucosa of the stomach, in which common microvessel characteristics were shown.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02367779
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  • 6
    Electronic Resource
    Electronic Resource
    Role of endogenous substance P in ethanol-induced mucosal damage in the rat stomach (1996)
    Springer
    Journal of gastroenterology 31 (1996), S. 314-322 
    Details
    ISSN: 1435-5922
    Keywords: rat stomach ; endogenous substance P ; capsaicin ; ethanol ; mucosal damage
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract To determine the role of endogenous substance P in ethanol-induced mucosal damage, two experiments were performed. In the first experiment, the stomachs of anesthetized rats were doubly cannulated and gastric damage was induced with 5 ml of 30% ethanol in the gastric lumen. The damage was ameliorated by pretreatment with capsaicin (0.16 and 1.6 mM) and spantide (100 mg/kg, i.v.). In the second experiment, the gastric mucosa of these rats was perfused with physiological saline containing pepstatin (10 μl/ml). Endogenous substance P (SP) in the perfusate was measured by enzyme immunoassay (EIA). The peak SP levels were increased by capsaicin (0.16–1.6 mM) in a concentration-dependent manner. Perfusion with 50% ethanol for 5 min increased the SP levels approximately threefold. Perfusion with 1.6 mM capsaicin, followed by 50% ethanol, reduced the injured area to about one-quarter of the original injured area. The peak SP levels during perfusion with 50% ethanol after pretreatment with 1.6 mM capsaicin did not differe from those observed after vehicle pretreatment (control). The area under the curve for SP release during 50% ethanol perfusion after vehicle perfusion was not reduced by previous perfusion with 1.6 mM capsaicin followed by 50% ethanol, indicating that the prevention of ethanol-induced injury by capsaicin may be due to excess amounts of different neuropeptides released simultaneously.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02355018
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  • 7
    Electronic Resource
    Electronic Resource
    Survival Probabilities for Discrete-Time Models in One Dimension (2000)
    Springer
    Journal of statistical physics 99 (2000), S. 603-612 
    Details
    ISSN: 1572-9613
    Keywords: survival probability ; the Domany–Kinzel model ; oriented percolation ; convergence theorem
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract We consider survival probabilities for the discrete-time process in one dimension, which is known as the Domany–Kinzel model. A convergence theorem for infinite systems can be obtained in the nonattractive case.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1018617328216
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  • 8
    Electronic Resource
    Electronic Resource
    Studies of four Japanese families with hereditary angioneurotic edema: Simultaneous activation of plasma protease systems and exogenous triggering stimuli (1984)
    Springer
    Annals of hematology 49 (1984), S. 405-418 
    Details
    ISSN: 1432-0584
    Keywords: Hereditary angioneurotic edema ; Complement ; Fibrinolysis ; Coagulation ; Kallikrein-kinin ; Exogenous trigger
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Forty-five relatives of 4 families with hereditary angioneurotic edema (HANE) were studied. Twenty-five, including 11 asymptomatic kindreds with the disposition, showed typical changes in complement system compatible with HANE. Follow-up study of HANE patients showed that, even in remission period, complement, coagulation and fibrinolytic systems can be activated. During edema attacks, moderate haemoconcentration and neutrophilia were encountered and kallikrein-kinin system was found to be also activated. Replacement therapy with C $$\bar l$$ -inhibitor preparation for an edema attack revealed that clinical improvement paralleled the increase in blood levels of high molecular weight kininogen. Thus, HANE attack is considered to be elicited in kindreds with the hereditary disposition by activation of plasma protease systems, particularly by that of kallikrein-kinin system. On the other hand, exogenous triggers that can initiate activation of the protease systems can be classified into 2, neuro-humoral (sympathetic nerve response) and physico-chemical, categories. Hence, the edema attack of kindreds with the hereditary disposition can at least be modified by the biosynthesis of plasma factors and the individual susceptibility to the liberated catecholamines. These two different reaction processes are considered to be linked by the release of plasminogen activator and/or Hageman factor activating enzyme.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00319889
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  • 9
    Electronic Resource
    Electronic Resource
    A possible role of prostaglandins and bradykinin as a trigger of exudation in carrageenin-induced rat pleurisy (1978)
    Springer
    Inflammation research 8 (1978), S. 108-112 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Pleurisy was induced in rats after intrapleural injection of 2% λ-carrageenin. The volume of the pleural exudate increased rapidly between 1 and 3 h, continuing to increase until 7 h. The dye amounts exuded into the pleural cavity for 20 min after 5% pontamine sky blue (60 mg/kg, i.v.) increased markedly at 1 to 3 h, then they kept constant until 6 h, decreasing thereafter. The PGE level in the pleural fluid increased rapidly during the period from 1 to 3 h, when the exudate commenced to accumulate, and then decreased slightly until 7 h. The main PG in the pleural fluid was recognized as PGE2 on thin-layer chromatography. The pretreatment of rats with a PG synthetase inhibitor, indomethacin (5 mg/kg, i.p.), 30 min before carrageenin administration resulted in the significant reduction of dye leakage at 1 h and of the pleural fluid exudation at 1 to 3 h. The intravenous injection into rats of stem bromelain (10 mg/kg, i.v.), a SH-protease from pineapples, caused the depletion of high molecular weight kininogen in plasma without effect on the content of low molecular weight kininogen. The pretreatment of rats with bromelain 30 min before carrageenin caused a marked reduction in the dye exudation during the periods from 1 to 3 h and 5 to 6 h. The accumulation of the exudate was strongly suppressed at the same periods. Although each treatment by itself did not completely abolish the dye and fluid exudation, the treatment of rats simultaneously with indomethacin and bromelain resulted in suppression of dye leakage from 1 to 6 h and practically no fluid accumulation was observed until 4 h. These results strongly indicate that prostaglandin is released in combination with bradykinin at the period when the pleural exudate commences to accumulate.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01972411
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  • 10
    Electronic Resource
    Electronic Resource
    An improved method for determination of the total kininogen in plasma (1978)
    Springer
    Inflammation research 8 (1978), S. 155-156 
    Details
    ISSN: 1420-908X
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01972422
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