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  • 1
    Electronic Resource
    Electronic Resource
    The Editor, ‘The British Journal of Dermatology’. Epidermolysis bullosa induced by penicillamine. (1967)
    Oxford, UK : Blackwell Publishing Ltd
    British journal of dermatology 79 (1967), S. 0 
    Details
    ISSN: 1365-2133
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2133.1967.tb11471.x
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  • 2
    Electronic Resource
    Electronic Resource
    The New Zealand rugby injury and performance project: alcohol use patterns within a cohort of rugby players (1996)
    Oxford, UK : Blackwell Publishing Ltd
    Addiction 91 (1996), S. 0 
    Details
    ISSN: 1360-0443
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine , Psychology
    Notes: Although heavy drinking has traditionally been associated with rugby, the drinking patterns of New Zealand rugby players have not been examined. Three hundred and forty-eight rugby players (257 males and 91 females), completed the Alcohol Use Disorders Identification Test (AUDIT) at the beginning of the 1993 rugby season. The mean AUDIT scores were 11.2 (SD = 5.1) for the males and 8.7 (4.1) for the females: a cut-off of 8.0 has been recommended by the developers of the instrument for identifying alcohol use disorders. The consumption of large quantities of alcohol, and a high frequency of heavy drinking sessions were the norm for this group. Sixty-one per cent of males and 38% of females consumed six or more drinks in a session at least weekly: typically, men drank 10 or more drinks in a session and women 5-6 drinks. The patterns of drinking exhibited by the cohort give cause for concern regarding the health risks associated with suck behaviour.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1046/j.1360-0443.1996.9112186510.x
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  • 3
    Electronic Resource
    Electronic Resource
    MELANOMA SPECIFIC PROTEIN: DETECTION AND CHARACTERIZATION OF A TUMOUR SPECIFIC PROTEIN FROM MELANOTIC URINE (1978)
    Oxford, UK : Blackwell Publishing Ltd
    Australasian journal of dermatology 19 (1978), S. 0 
    Details
    ISSN: 1440-0960
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Medicine
    Notes: Urine from patients with malignant melanoma contains a specific protein not detected in normal urine nor in the urine of any other cancer patients tested. This protein is also found in urine from patients with actively developing halo naevi. It is detected by its ability to inhibit the cytoplasmic staining of malignant melanoma cells by rabbit antimelanoma antisera. The protein is sialo-protein, molecular weight 76,000 daltons, with α1 globulin mobility. It is suggested that it may be related to the melanosomal proteins.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1440-0960.1978.tb00180.x
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  • 4
    Electronic Resource
    Electronic Resource
    Radiologic-Pathologic Conference of Children's Hospital Boston: New pulmonary nodules in a child undergoing treatment for a solid malignancy (1996)
    Springer
    Pediatric radiology 26 (1996), S. 19-21 
    Details
    ISSN: 1432-1998
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract The appearance of pulmonary nodules in a child with a solid malignancy undergoing chemotherapy is a clinically worrisome event. The diagnosis in such cases is not always metastatic disease, and the differential diagnosis should include granulomatous disease, atelectasis, pneumonia, inflammatory pseudotumor, hamartoma, radiation pneumonitis, and bronchiolitis obliterans with organizing pneumonia. There is no consistent radiologic feature to help distinguish benign from malignant causes of these new lesions. However, repeat chest CT 4–6 weeks after the lesions are first noted can be used to track lesion progression and may obviate the need for biopsy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01403697
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  • 5
    Electronic Resource
    Electronic Resource
    Analysis of an SEIRS epidemic model with two delays (1996)
    Springer
    Journal of mathematical biology 35 (1996), S. 240-260 
    Details
    ISSN: 1432-1416
    Keywords: Key words: Epidemic model ; Integro-differential equation ; Delay equation ; Epidemic threshold
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Mathematics
    Notes: Abstract.  A disease transmission model of SEIRS type with exponential demographic structure is formulated. All newborns are assumed susceptible, there is a natural death rate constant, and an excess death rate constant for infective individuals. Latent and immune periods are assumed to be constants, and the force of infection is assumed to be of the standard form, namely proportional to I(t)/N(t) where N(t) is the total (variable) population size and I(t) is the size of the infective population. The model consists of a set of integro-differential equations. Stability of the disease free proportion equilibrium, and existence, uniqueness, and stability of an endemic proportion equilibrium, are investigated. The stability results are stated in terms of a key threshold parameter. More detailed analyses are given for two cases, the SEIS model (with no immune period), and the SIRS model (with no latent period). Several threshold parameters quantify the two ways that the disease can be controlled, by forcing the number or the proportion of infectives to zero.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002850050051
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  • 6
    Electronic Resource
    Electronic Resource
    On the role of long incubation periods in the dynamics of acquired immunodeficiency syndrome (AIDS) (1989)
    Springer
    Journal of mathematical biology 27 (1989), S. 373-398 
    Details
    ISSN: 1432-1416
    Keywords: Acquired immunodeficiency syndrome (AIDS) ; Distributed delays ; Human immunodeficiency virus (HIV) ; Sexually transmitted diseases
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Mathematics
    Notes: Abstract In this study, we investigate systematically the role played by the reproductive number (the number of secondary infections generated by an infectious individual in a population of susceptibles) on single group populations models of the spread of HIV/AIDS. Our results for a single group model show that if R ⩽ 1, the disease will die out, and strongly suggest that if R 〉 1 the disease will persist regardless of initial conditions. Our extensive (but incomplete) mathematical analysis and the numerical simulations of various research groups support the conclusion that the reproductive number R is a global bifurcation parameter. The bifurcation that takes place as R is varied is a transcritical bifurcation; in other words, when R crosses 1 there is a global transfer of stability from the infection-free state to the endemic equilibrium, and vice versa. These results do not depend on the distribution of times spent in the infectious categories (the survivorship functions). Furthermore, by keeping all the key statistics fixed, we can compare two extremes: exponential survivorship versus piecewise constant survivorship (individuals remain infectious for a fixed length of time). By choosing some realistic parameters we can see (at least in these cases) that the reproductive numbers corresponding to these two extreme cases do not differ significantly whenever the two distributions have the same mean. At any rate a formula is provided that allows us to estimate the role played by the survivorship function (and hence the incubation period) in the global dynamics of HIV. These results support the conclusion that single population models of this type are robust and hence are good building blocks for the construction of multiple group models. Our understanding of the dynamics of HIV in the context of mathematical models for multiple groups is critical to our understanding of the dynamics of HIV in a highly heterogeneous population.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00290636
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  • 7
    Electronic Resource
    Electronic Resource
    Analysis of a model of a vertically transmitted disease (1983)
    Springer
    Journal of mathematical biology 17 (1983), S. 305-329 
    Details
    ISSN: 1432-1416
    Keywords: Epidemic models ; Vertically transmitted diseases ; Maturation periods ; Spatial diffusion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Mathematics
    Notes: Abstract A model is presented of a disease that can be transmitted directly from parent to offspring (vertical transmission) as well as through contact with infectives. A global stability analysis is given for the basic model and the epidemiological effects of vertical transmission are discussed. The effects of the addition of maturation and incubation delays as well as spatial diffusion are analyzed in some special cases.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00276519
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  • 8
    Electronic Resource
    Electronic Resource
    Interaction of maturation delay and nonlinear birth in population and epidemic models (1999)
    Springer
    Journal of mathematical biology 39 (1999), S. 332-352 
    Details
    ISSN: 1432-1416
    Keywords: Key words: Maturation delay ; Epidemic model ; Global stability ; Periodic solutions
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Mathematics
    Notes: Abstract.  A population with birth rate function B(N) N and linear death rate for the adult stage is assumed to have a maturation delay T〉0. Thus the growth equation N′(t)=B(N(t−T)) N(t−T) e− d 1 T−dN(t) governs the adult population, with the death rate in previous life stages d 1≧0. Standard assumptions are made on B(N) so that a unique equilibrium N e exists. When B(N) N is not monotone, the delay T can qualitatively change the dynamics. For some fixed values of the parameters with d 1〉0, as T increases the equilibrium N e can switch from being stable to unstable (with numerically observed periodic solutions) and then back to stable. When disease that does not cause death is introduced into the population, a threshold parameter R 0 is identified. When R 0〈1, the disease dies out; when R 0〉1, the disease remains endemic, either tending to an equilibrium value or oscillating about this value. Numerical simulations indicate that oscillations can also be induced by disease related death in a model with maturation delay.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002850050194
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  • 9
    Electronic Resource
    Electronic Resource
    Kinetics and mechanisms of the reactions of OH with some oxygenated compounds of importance in tropospheric chemistry (1994)
    New York, NY : Wiley-Blackwell
    International Journal of Chemical Kinetics 26 (1994), S. 113-130 
    Details
    ISSN: 0538-8066
    Keywords: Chemistry ; Physical Chemistry
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Notes: The kinetics of the reactions have been studied in a discharge flow system under pseudo-first-order conditions. The OH concentration was monitored by laser induced fluorescence and helium was used as the carrier gas. Values of k1 = (8.1 ± 1.7) × 10-13, k2 = (1.31 ± 0.26) × 10-11, k3 = (2.6 ± 0.5) × 10-11, and k4 = (2.5 ± 0.4) × 10-11 cm3 molecule-1 s-1, at 298 K and 1 torr total pressure, were obtained.To validate the newly constructed system the rate constant for the reaction was determined in a similar manner. The value of k5 = (6.7 ± 0.9) × 10-12 cm3 molecule-1 s-1 at 298 K and 1 torr total pressure is in very good agreement with other literature values.The mechanisms for the atmospheric degradation of these compounds have been constructed to allow their incorporation in a photochemical trajectory computer model, to assess their impact on photochemical ozone creation in the troposphere. © 1994 John Wiley & Sons, Inc.
    Additional Material: 3 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/kin.550260112
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  • 10
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    IL-22 Regulates Iron Availability In Vivo through the Induction of Hepcidin [INNATE IMMUNITY AND INFLAMMATION] (2013)
    The American Association of Immunologists (AAI)
    Details
    Publication Date: 2013-08-03
    Description: Iron is a trace element important for the proper folding and function of various proteins. Physiological regulation of iron stores is of critical importance for RBC production and antimicrobial defense. Hepcidin is a key regulator of iron levels within the body. Under conditions of iron deficiency, hepcidin expression is reduced to promote increased iron uptake from the diet and release from cells, whereas during conditions of iron excess, induction of hepcidin restricts iron uptake and movement within the body. The cytokine IL-6 is well established as an important inducer of hepcidin. The presence of this cytokine during inflammatory states can induce hepcidin production, iron deficiency, and anemia. In this study, we show that IL-22 also influences hepcidin production in vivo. Injection of mice with exogenous mouse IgG1 Fc fused to the N terminus of mouse IL-22 (Fc–IL-22), an IL-22R agonist with prolonged and enhanced functional potency, induced hepcidin production, with a subsequent decrease in circulating serum iron and hemoglobin levels and a concomitant increase in iron accumulation within the spleen. This response was independent of IL-6 and was attenuated in the absence of the IL-22R–associated signaling kinase, Tyk2. Ab-mediated blockade of hepcidin partially reversed the effects on iron biology caused by IL-22R stimulation. Taken together, these data suggest that exogenous IL-22 regulates hepcidin production to physiologically influence iron usage.
    Print ISSN: 0022-1767
    Electronic ISSN: 1550-6606
    Topics: Medicine
    Published by The American Association of Immunologists (AAI)
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