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  • 1
    Online Resource
    Online Resource
    Discovering Finance Keywords via Continuous-Space Language Models
    Association for Computing Machinery (ACM) ; 2016
    In:  ACM Transactions on Management Information Systems Vol. 7, No. 3 ( 2016-10-18), p. 1-17
    Details
    In: ACM Transactions on Management Information Systems, Association for Computing Machinery (ACM), Vol. 7, No. 3 ( 2016-10-18), p. 1-17
    Abstract: The growing amount of public financial data makes it increasingly important to learn how to discover valuable information for financial decision making. This article proposes an approach to discovering financial keywords from a large number of financial reports. In particular, we apply the continuous bag-of-words (CBOW) model, a well-known continuous-space language model, to the textual information in 10-K financial reports to discover new finance keywords. In order to capture word meanings to better locate financial terms, we also present a novel technique to incorporate syntactic information into the CBOW model. Experimental results on four prediction tasks using the discovered keywords demonstrate that our approach is effective for discovering predictability keywords for post-event volatility, stock volatility, abnormal trading volume, and excess return predictions. We also analyze the discovered keywords that attest to the ability of the proposed method to capture both syntactic and contextual information between words. This shows the success of this method when applied to the field of finance.
    Type of Medium: Online Resource
    ISSN: 2158-656X , 2158-6578
    URL: Article
    DOI: 10.1145/2948072
    Language: English
    Publisher: Association for Computing Machinery (ACM)
    Publication Date: 2016
    detail.hit.zdb_id: 2593589-6
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  • 2
    Online Resource
    Online Resource
    Crystal Structure and Acid-Base Properties of Ozagrel
    Acta Physico-Chimica Sinica & University Chemistry Editorial Office, Peking University ; 2008
    In:  Acta Physico-Chimica Sinica Vol. 24, No. 01 ( 2008), p. 176-178
    Details
    In: Acta Physico-Chimica Sinica, Acta Physico-Chimica Sinica & University Chemistry Editorial Office, Peking University, Vol. 24, No. 01 ( 2008), p. 176-178
    Type of Medium: Online Resource
    ISSN: 1000-6818
    URL: Article
    DOI: 10.3866/PKU.WHXB20080132
    Language: English
    Publisher: Acta Physico-Chimica Sinica & University Chemistry Editorial Office, Peking University
    Publication Date: 2008
    detail.hit.zdb_id: 2474339-2
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  • 3
    Online Resource
    Online Resource
    A Closed-Form Formula for an Option with Discrete and Continuous Barriers
    Informa UK Limited ; 2010
    In:  Communications in Statistics - Theory and Methods Vol. 40, No. 2 ( 2010-12-06), p. 345-357
    Details
    In: Communications in Statistics - Theory and Methods, Informa UK Limited, Vol. 40, No. 2 ( 2010-12-06), p. 345-357
    Type of Medium: Online Resource
    ISSN: 0361-0926 , 1532-415X
    URL: Article
    DOI: 10.1080/03610920903402605
    Language: English
    Publisher: Informa UK Limited
    Publication Date: 2010
    detail.hit.zdb_id: 1476983-9
    detail.hit.zdb_id: 283673-7
    SSG: 11
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  • 4
    Online Resource
    Online Resource
    ADAMTS-7 Mediates Vascular Smooth Muscle Cell Migration and Neointima Formation in Balloon-Injured Rat Arteries
    Ovid Technologies (Wolters Kluwer Health) ; 2009
    In:  Circulation Research Vol. 104, No. 5 ( 2009-03-13), p. 688-698
    Details
    In: Circulation Research, Ovid Technologies (Wolters Kluwer Health), Vol. 104, No. 5 ( 2009-03-13), p. 688-698
    Abstract: The migration of vascular smooth muscle cells (VSMCs) plays an essential role during the development of atherosclerosis and restenosis. Extensive studies have implicated the importance of extracellular matrix (ECM)-degrading proteinases in VSMC migration. A recently described family of proteinases, a disintegrin and metalloproteinase with thrombospondin motifs (ADAMTs), is capable of degrading vascular ECM proteins. Here, we sought to determine whether ADAMTS-7 is involved in VSMC migration and neointima formation in response to vascular injury. ADAMTS-7 protein accumulated preferentially in neointima of the carotid artery wall after balloon injury. In primary VSMCs, ADAMTS-7 level was enhanced by the proinflammatory cytokine tumor necrosis factor α and growth factor platelet-derived growth factor-BB. ADAMTS-7 overexpression greatly accelerated and small interfering RNA knockdown markedly retarded VSMC migration/invasion in vitro. In addition, luminal delivery of ADAMTS-7 adenovirus to carotid arteries exacerbated intimal thickening nearly sixfold 7 days after injury. Conversely, perivascular administration of ADAMTS-7 small interfering RNA but not scramble small interfering RNA to injured arteries attenuated intimal thickening by 50% at 14 days after injury. Furthermore, ADAMTS-7 mediated degradation of the vascular ECM cartilage oligomeric matrix protein (COMP) in injured vessels. Replenishing COMP circumvented the promigratory effect of ADAMTS-7 on VSMCs. Enforced expression of COMP significantly suppressed VSMC migration and neointima formation postinjury, which indicates that ADAMTS-7 facilitated intimal hyperplasia through degradation of inhibitory matrix protein COMP. ADAMTS-7 may therefore serve as a novel therapeutic target for atherosclerosis and postangioplasty restenosis.
    Type of Medium: Online Resource
    ISSN: 0009-7330 , 1524-4571
    URL: Article
    DOI: 10.1161/CIRCRESAHA.108.188425
    RVK:
    XA 10000
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 2009
    detail.hit.zdb_id: 1467838-X
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  • 5
    Online Resource
    Online Resource
    Cartilage Oligomeric Matrix Protein Inhibits Vascular Smooth Muscle Calcification by Interacting With Bone Morphogenetic Protein-2
    Ovid Technologies (Wolters Kluwer Health) ; 2011
    In:  Circulation Research Vol. 108, No. 8 ( 2011-04-15), p. 917-928
    Details
    In: Circulation Research, Ovid Technologies (Wolters Kluwer Health), Vol. 108, No. 8 ( 2011-04-15), p. 917-928
    Abstract: Vascular calcification is a significant contributor to cardiovascular morbidity and mortality. We recently reported that cartilage oligomeric matrix protein (COMP) is pivotal for maintaining the homeostasis of vascular smooth muscle cells (VSMCs). Whether COMP affects the process of vascular calcification is unknown. Objective: We aimed to test whether COMP modulates vascular calcification. Methods and Results: VSMC calcification in vitro was induced by calcifying media containing high inorganic phosphate or calcium. In vivo medial vessel calcification was induced in rats by 5/6 nephrectomy with a high-phosphate diet or by periadventitial application of CaCl 2 to the abdominal aorta. COMP protein level was markedly reduced in both calcified VSMCs and arteries. COMP deficiency remarkably exacerbated VSMC calcification, whereas ectopic expression of COMP greatly reduced calcification. Furthermore, COMP knockdown facilitated osteogenic markers expression by VSMCs even in the absence of calcifying media. By contrast, COMP overexpression significantly inhibited high phosphate– or high calcium–induced VSMC osteochondrogenic transition. Induction of osteogenic marker expression by COMP silencing was reversed by a soluble form of bone morphogenetic protein (BMP)-2 receptor IA, which suggests a BMP-2–dependent mechanism. Our data revealed that COMP bound directly to BMP-2 through the C terminus, inhibited BMP-2 receptor binding, and blocked BMP-2 osteogenic signaling, indicating COMP inhibits osteochondrogenic transition of VSMCs at least partially through inhibiting BMP-2. Conclusions: Our data strongly suggest that COMP is a novel inhibitor of vascular calcification. The imbalance between the effects of COMP and BMP-2 may provide new insights into the pathophysiology of vascular calcification.
    Type of Medium: Online Resource
    ISSN: 0009-7330 , 1524-4571
    URL: Article
    DOI: 10.1161/CIRCRESAHA.110.234328
    RVK:
    XA 10000
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 2011
    detail.hit.zdb_id: 1467838-X
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  • 6
    Online Resource
    Online Resource
    UGSD: User Generated Sentiment Dictionaries from Online Customer Reviews
    Association for the Advancement of Artificial Intelligence (AAAI) ; 2019
    In:  Proceedings of the AAAI Conference on Artificial Intelligence Vol. 33, No. 01 ( 2019-07-17), p. 313-320
    Details
    In: Proceedings of the AAAI Conference on Artificial Intelligence, Association for the Advancement of Artificial Intelligence (AAAI), Vol. 33, No. 01 ( 2019-07-17), p. 313-320
    Abstract: Customer reviews on platforms such as TripAdvisor and Amazon provide rich information about the ways that people convey sentiment on certain domains. Given these kinds of user reviews, this paper proposes UGSD, a representation learning framework for constructing domain-specific sentiment dictionaries from online customer reviews, in which we leverage the relationship between user-generated reviews and the ratings of the reviews to associate the reviewer sentiment with certain entities. The proposed framework has the following three main advantages. First, no additional annotations of words or external dictionaries are needed for the proposed framework; the only resources needed are the review texts and entity ratings. Second, the framework is applicable across a variety of user-generated content from different domains to construct domain-specific sentiment dictionaries. Finally, each word in the constructed dictionary is associated with a low-dimensional dense representation and a degree of relatedness to a certain rating, which enable us to obtain more fine-grained dictionaries and enhance the application scalability of the constructed dictionaries as the word representations can be adopted for various tasks or applications, such as entity ranking and dictionary expansion. The experimental results on three real-world datasets show that the framework is effective in constructing high-quality domain-specific sentiment dictionaries from customer reviews.
    Type of Medium: Online Resource
    ISSN: 2374-3468 , 2159-5399
    URL: Article
    DOI: 10.1609/aaai.v33i01.3301313
    Language: Unknown
    Publisher: Association for the Advancement of Artificial Intelligence (AAAI)
    Publication Date: 2019
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    Online Resource
  • 7
    Online Resource
    Online Resource
    Cartilage Oligomeric Matrix Protein Maintains the Contractile Phenotype of Vascular Smooth Muscle Cells by Interacting With α 7 β 1 Integrin
    Ovid Technologies (Wolters Kluwer Health) ; 2010
    In:  Circulation Research Vol. 106, No. 3 ( 2010-02-19), p. 514-525
    Details
    In: Circulation Research, Ovid Technologies (Wolters Kluwer Health), Vol. 106, No. 3 ( 2010-02-19), p. 514-525
    Abstract: Rational : Vascular smooth muscle cells (VSMCs) switching from a contractile/differentiated to a synthetic/dedifferentiated phenotype has an essential role in atherosclerosis, postangioplastic restenosis and hypertension. However, how normal VSMCs maintain the differentiated state is less understood. Objective : We aimed to indentify the effect of cartilage oligomeric matrix protein (COMP), a normal vascular extracellular matrix, on modulation of VSMCs phenotype. Methods and Results : We demonstrated that COMP was associated positively with the expression of VSMC differentiation marker genes during phenotype transition. Knockdown of COMP by small interfering (si)RNA favored dedifferentiation. Conversely, adenoviral overexpression of COMP markedly suppressed platelet-derived growth factor-BB-elicited VSMC dedifferentiation, characterized by altered VSMC morphology, actin fiber organization, focal adhesion assembly, and the expression of phenotype-dependent markers. Whereas α 7 integrin coimmunoprecipitated with COMP in normal rat VSMCs and vessels, neutralizing antibody or siRNA against α 7 integrin inhibited VSMC adhesion to COMP, which indicated that α 7 β 1 integrin is a potential receptor for COMP. As well, blocking or interference by siRNA of α 7 integrin completely abolished the effect of COMP on conserving the contractile phenotype. In accordance, ectopic adenoviral overexpression of COMP greatly retarded VSMC phenotype switching, rescued contractility of carotid artery ring, and inhibited neointima formation in balloon-injured rats. Conclusions : Our data suggest that COMP is essential for maintaining a VSMC contractile phenotype and the protective effects of COMP are mainly mediated through interaction with α 7 β 1 integrin. Investigations to identify the factors affecting the expression and integrity of COMP may provide a novel therapeutic target for vascular disorders.
    Type of Medium: Online Resource
    ISSN: 0009-7330 , 1524-4571
    URL: Article
    DOI: 10.1161/CIRCRESAHA.109.202762
    RVK:
    XA 10000
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 2010
    detail.hit.zdb_id: 1467838-X
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  • 8
    Online Resource
    Online Resource
    P4‐274: Does Mmse Exactly Work For Diagnosing Alzheimer's Disease
    Wiley ; 2009
    In:  Alzheimer's & Dementia Vol. 5, No. 4S_Part_17 ( 2009-07)
    Details
    In: Alzheimer's & Dementia, Wiley, Vol. 5, No. 4S_Part_17 ( 2009-07)
    Type of Medium: Online Resource
    ISSN: 1552-5260 , 1552-5279
    URL: Article
    DOI: 10.1016/j.jalz.2009.07.055
    Language: English
    Publisher: Wiley
    Publication Date: 2009
    detail.hit.zdb_id: 2201940-6
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  • 9
    Online Resource
    Online Resource
    A stochastic‐volatility equity‐price tree for pricing convertible bonds with endogenous firm values and default risks determined by the first‐passage default model
    Wiley ; 2022
    In:  Journal of Futures Markets Vol. 42, No. 12 ( 2022-12), p. 2103-2134
    Details
    In: Journal of Futures Markets, Wiley, Vol. 42, No. 12 ( 2022-12), p. 2103-2134
    Abstract: This paper proposes a novel equity‐price‐tree‐based convertible bond (CB) pricing model based on the first‐passage default model under stochastic interest rates. By regarding equity values as down‐and‐out call options on firm values (FVs), at each tree node, we solve the implied FV and equity‐price volatility (EPV), and then endogenously settle the default probability (DP) and also the dilution effect subject to CB conversions with the implied FV and capital structure. Our model captures the stylized negative (positive) relationships between the stochastically evolving DP and FV or EP (EPV) that cannot be fully achieved by existing CB pricing models.
    Type of Medium: Online Resource
    ISSN: 0270-7314 , 1096-9934
    URL: Issue
    URL: Article
    DOI: 10.1002/fut.v42.12
    DOI: 10.1002/fut.22370
    Language: English
    Publisher: Wiley
    Publication Date: 2022
    detail.hit.zdb_id: 2002201-3
    SSG: 3,2
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  • 10
    Online Resource
    Online Resource
    Efficient and Robust Combinatorial Option Pricing Algorithms on the Trinomial Lattice for Polynomial and Barrier Options
    Hindawi Limited ; 2022
    In:  Mathematical Problems in Engineering Vol. 2022 ( 2022-5-21), p. 1-20
    Details
    In: Mathematical Problems in Engineering, Hindawi Limited, Vol. 2022 ( 2022-5-21), p. 1-20
    Abstract: Options can be priced by the lattice model, the results of which converge to the theoretical option value as the lattice’s number of time steps n approaches infinity. The time complexity of a common dynamic programming pricing approach on the lattice is slow (at least O n 2 ), and a large n is required to obtain accurate option values. Although O n -time combinatorial pricing algorithms have been developed for the classical binomial lattice, significantly oscillating convergence behavior makes them impractical. The flexibility of trinomial lattices can be leveraged to reduce the oscillation, but there are as yet no linear-time algorithms on trinomial lattices. We develop O n -time combinatorial pricing algorithms for polynomial options that cannot be analytically priced. The commonly traded plain vanilla and power options are degenerated cases of polynomial options. Barrier options that cannot be stably priced by the binomial lattice can be stably priced by our O n -time algorithm on a trinomial lattice. Numerical experiments demonstrate the efficiency and accuracy of our O n -time trinomial lattice algorithms.
    Type of Medium: Online Resource
    ISSN: 1563-5147 , 1024-123X
    URL: Article
    DOI: 10.1155/2022/5843491
    Language: English
    Publisher: Hindawi Limited
    Publication Date: 2022
    detail.hit.zdb_id: 2014442-8
    SSG: 11
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