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  • PACS. 75.30.Vn Colossal magnetoresistance – 75.30.Kz Magnetic phase boundaries (including magnetic transitions, metamagnetism, etc.) – 75.25.+z Spin arrangements in magnetically ordered materials (including neutron and spin-polarized electron studies, synchrotron-source X-ray scattering, etc.)  (1)
  • Risk Factors, Genetic, Association Studies, Ischemic Stroke  (1)
  • 1
    Electronic Resource
    Electronic Resource
    Effect of Cu doping at the Mn site on the transport aaand magnetic behaviors of La 2/3 Ca 1/3 MnO3 (2001)
    Springer
    The European physical journal 20 (2001), S. 177-181 
    Details
    ISSN: 1434-6036
    Keywords: PACS. 75.30.Vn Colossal magnetoresistance – 75.30.Kz Magnetic phase boundaries (including magnetic transitions, metamagnetism, etc.) – 75.25.+z Spin arrangements in magnetically ordered materials (including neutron and spin-polarized electron studies, synchrotron-source X-ray scattering, etc.)
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract: We show that the replacement of Mn with Cu, without any structure change, strongly affects the transport and magnetic behaviors in La 2/3 Ca 1/3 Mn1-xCuxO 3 - δ (x ?0.3). Doping largely shifts the insulator-metal transition towards the low-temperature regime and causes substantial enhancements in both resistance and magnetoresistance near the transition for doping range of x ? 15%, and further doping to x ≥ 20% shows insulating behavior. For 15% doping, the sample shows large magnetoresistances (∼104%) over the whole low temperature range. Magnetization measurement at helium temperature reveals that samples for x ? 10% and ≥ 20% basically show ferromagnetic (FM) and antiferromagnetic (AF) behaviors, respectively, while the 15% Cu doped sample undergoes a metamagnetic transition from AF state to FM state on increasing field. We argue that doping introduces the AF insulating phase into the system, causing unusual transport and magnetic behaviors.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01315688
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    Paper (German National Licenses)
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  • 2
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    Homozygous ALDH2*2 Is an Independent Risk Factor for Ischemic Stroke in Taiwanese Men [Clinical Sciences] (2016)
    American Heart Association (AHA)
    In: Stroke
    Details
    Publication Date: 2016-08-23
    Description: Background and Purpose— The * 2 allele of the aldehyde dehydrogenase 2 gene (ALDH2 ) is the most common variant in Asian populations. The variant resulting in enzyme dysfunction was highly related to coronary artery disease. Recently, genome-wide association studies also discovered that the 12q24 locus near ALDH2 gene was associated with hypertension and ischemic stroke. This study intended to further investigate whether the above variant of ALDH2 increases the risk for ischemic stroke in Taiwanese. Methods— A case–control study was conducted on 914 patients with acute ischemic stroke and 746 nonstroke controls. Polymerase chain reaction and sequencing were used to identify the ALDH2 genotype. Vascular risk factors, stroke subtypes, vascular stenosis, and stroke outcomes were analyzed. Results— ALDH2 genotypes differed significantly between male controls (* 1/*1 versus * 1/*2 versus * 2/*2 =53.8% versus 39.9% versus 6.4%) and male patients with ischemic stroke (* 1/*1 versus * 1/*2 versus * 2/*2 =51.5% versus 37.3% versus 11.2%; P =0.048). No significant difference was found between groups for female patients ( P =0.228). Multivariate logistic regression analysis revealed that the ALDH2*2/*2 genotype was an independent risk factor for ischemic stroke in male patients (odds ratio, 1.93 [95% confidence interval, 1.07–3.46]; P =0.028). Further analysis of men with ischemic stroke demonstrated that the polymorphism of ALDH2 was not related to vascular risk factors, severity of vascular atherosclerosis, stroke subtypes, and stroke functional outcomes. Conclusions— The study demonstrated that ALDH2*2/*2 may be an independent risk factor for ischemic stroke in Taiwanese men, but not in Taiwanese women.
    Keywords: Risk Factors, Genetic, Association Studies, Ischemic Stroke
    Print ISSN: 0039-2499
    Electronic ISSN: 1524-4628
    Topics: Medicine
    Published by American Heart Association (AHA)
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