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  • 1
    Online Resource
    Online Resource
    Cham :Springer International Publishing AG,
    Keywords: Neurosciences. ; Electronic books.
    Type of Medium: Online Resource
    Pages: 1 online resource (180 pages)
    Edition: 1st ed.
    ISBN: 9783319553108
    Series Statement: Intelligent Systems Reference Library ; v.126
    DDC: 006.3
    Language: English
    Note: Intro -- Preface -- Contents -- Acronyms -- 1 Introduction -- 1.1 Background -- 1.2 Spiking Neurons -- 1.2.1 Biological Background -- 1.2.2 Generations of Neuron Models -- 1.2.3 Spiking Neuron Models -- 1.3 Neural Codes -- 1.3.1 Rate Code -- 1.3.2 Temporal Code -- 1.3.3 Temporal Code Versus Rate Code -- 1.4 Cognitive Learning and Memory in the Brain -- 1.4.1 Temporal Learning -- 1.4.2 Cognitive Memory in the Brain -- 1.5 Objectives and Contributions -- 1.6 Outline of the Book -- References -- 2 Rapid Feedforward Computation by Temporal Encoding and Learning with Spiking Neurons -- 2.1 Introduction -- 2.2 The Spiking Neural Network -- 2.3 Single-Spike Temporal Coding -- 2.4 Temporal Learning Rule -- 2.4.1 The Tempotron Rule -- 2.4.2 The ReSuMe Rule -- 2.4.3 The Tempotron-Like ReSuMe Rule -- 2.5 Simulation Results -- 2.5.1 The Data Set and the Classification Problem -- 2.5.2 Encoding Images -- 2.5.3 Choosing Among Temporal Learning Rules -- 2.5.4 The Properties of Tempotron Rule -- 2.5.5 Recognition Performance -- 2.6 Discussion -- 2.6.1 Encoding Benefits from Biology -- 2.6.2 Types of Synapses -- 2.6.3 Schemes of Readout -- 2.6.4 Extension of the Network for Robust Sound Recognition -- 2.7 Conclusion -- References -- 3 A Spike-Timing Based Integrated Model for Pattern Recognition -- 3.1 Introduction -- 3.2 The Integrated Model -- 3.2.1 Neuron Model and General Structure -- 3.2.2 Latency-Phase Encoding -- 3.2.3 Supervised Spike-Timing Based Learning -- 3.3 Numerical Simulations -- 3.3.1 Network Architecture and Encoding of Grayscale Images -- 3.3.2 Learning Performance -- 3.3.3 Generalization Capability -- 3.3.4 Parameters Evaluation -- 3.3.5 Capacity of the Integrated System -- 3.4 Related Works -- 3.5 Conclusions -- References -- 4 Precise-Spike-Driven Synaptic Plasticity for Hetero Association of Spatiotemporal Spike Patterns. , 4.1 Introduction -- 4.2 Methods -- 4.2.1 Spiking Neuron Model -- 4.2.2 PSD Learning Rule -- 4.3 Results -- 4.3.1 Association of Single-Spike and Multi-spike Patterns -- 4.3.2 Generality to Different Neuron Models -- 4.3.3 Robustness to Noise -- 4.3.4 Learning Capacity -- 4.3.5 Effects of Learning Parameters -- 4.3.6 Classification of Spatiotemporal Patterns -- 4.4 Discussion and Conclusion -- References -- 5 A Spiking Neural Network System for Robust Sequence Recognition -- 5.1 Introduction -- 5.2 The Integrated Network for Sequence Recognition -- 5.2.1 Rationale of the Whole System -- 5.2.2 Neural Encoding Method -- 5.2.3 Item Recognition with the PSD Rule -- 5.2.4 The Spike Sequence Decoding Method -- 5.3 Experimental Results -- 5.3.1 Learning Performance Analysis of the PSD Rule -- 5.3.2 Item Recognition -- 5.3.3 Spike Sequence Decoding -- 5.3.4 Sequence Recognition System -- 5.4 Discussions -- 5.4.1 Temporal Learning Rules and Spiking Neurons -- 5.4.2 Spike Sequence Decoding Network -- 5.4.3 Potential Applications in Authentication -- 5.5 Conclusion -- References -- 6 Temporal Learning in Multilayer Spiking Neural Networks Through Construction of Causal Connections -- 6.1 Introduction -- 6.2 Multilayer Learning Rules -- 6.2.1 Spiking Neuron Model -- 6.2.2 Multilayer PSD Rule -- 6.2.3 Multilayer Tempotron Rule -- 6.3 Heuristic Discussion on the Multilayer Learning Rules -- 6.4 Simulation Results -- 6.4.1 Construction of Causal Connections -- 6.4.2 The XOR Benchmark -- 6.4.3 The Iris Benchmark -- 6.5 Discussion and Conclusion -- References -- 7 A Hierarchically Organized Memory Model with Temporal Population Coding -- 7.1 Introduction -- 7.2 The Hierarchical Organized Memory Model -- 7.2.1 Neuron Models and Neural Oscillations -- 7.2.2 Temporal Population Coding -- 7.2.3 The Tempotron Learning and STDP -- 7.3 Simulation Results. , 7.3.1 Auto-Associative Memory -- 7.3.2 Episodic Memory -- 7.4 Discussion -- 7.4.1 Information Flow and Emergence of Neural Cliques -- 7.4.2 Storage, Recall and Organization of Memory -- 7.4.3 Temporal Compression and Information Binding -- 7.4.4 Related Works -- 7.5 Conclusion -- References -- 8 Spiking Neuron Based Cognitive Memory Model -- 8.1 Introduction -- 8.2 SRM-Based CA3 Model -- 8.2.1 Spike Response Model -- 8.2.2 SRM-Based Pyramidal Cell -- 8.2.3 SRM-Based Interneuron -- 8.3 Convergence of Synaptic Weight -- 8.4 Maximum Synaptic Weight to Prevent Early Activation -- 8.5 Pattern Completion of Auto-Associative Memory -- 8.6 Discussion -- 8.7 Conclusion -- References.
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  • 2
    ISSN: 1432-0878
    Keywords: Key words: Enteric nervous system ; NK3 receptor ; Sensory neurons ; Co-localisation ; NK1 receptor ; Calcium-binding proteins ; Nitric oxide synthase
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. The localisation of the neurokinin 3 receptor (NK3r) in the rat gastrointestinal tract has been studied by using a polyclonal antiserum against the C-terminal portion (amino acids 388–452) of the rat NK3r. In the oesophagus, immunoreactivity for the NK3r was found on smooth muscle cells of the muscularis mucosae. NK3r immunoreactivity was not present on muscle cells of other regions. Nerve cell bodies immunoreactive for NK3r were seen in the myenteric and submucous plexuses of the small and large intestine, but not in the stomach or oesophagus. Immunoreactivity was largely confined to nerve cell surfaces. The reaction product was on the cell soma and initial parts of axons. Reactivity was not seen on nerve terminals. Immunoreactive nerve cells had Dogiel Type II morphology. Patterns of co-localisation of NK3r and immunoreactivity for other markers were examined in the ileum, to provide a basis from which to deduce the functional identity of NK3r-immunoreactive nerve cells. Most of the NK3r-immunoreactive nerve cells were also immunoreactive for the calcium-binding proteins, calretinin and calbindin, and all were immunoreactive for the NK1 receptor (NK1r). Nerve cells that were immunoreactive for nitric oxide synthase were not immunoreactive for either NK3r or NK1r. The projections of the calbindin and calretinin neurons were determined by nerve lesion studies. Their morphology, projections to the mucosa and other ganglia and immunoreactivity for the calcium-binding proteins suggest that the NK3r-immunoreactive neurons are intrinsic sensory neurons.
    Type of Medium: Electronic Resource
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