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  • muscle  (4)
  • Key words:Biochemical markers – Distal forearm fracture – Dual-energy X-ray absorptiometry – Reflex sympathetic dystrophy – Speed of sound  (1)
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  • 1
    Electronic Resource
    Electronic Resource
    Changes in Bone Mass and Bone Turnover Following Distal Forearm Fracture (1999)
    Springer
    Osteoporosis international 10 (1999), S. 399-407 
    Details
    ISSN: 1433-2965
    Keywords: Key words:Biochemical markers – Distal forearm fracture – Dual-energy X-ray absorptiometry – Reflex sympathetic dystrophy – Speed of sound
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract: Bone loss occurs close to a fracture and is associated with increased bone turnover. Fracture healing itself results in increased markers of bone turnover. But the exact patterns of these changes after different fractures are unclear. We aimed to investigate the changes in bone density and biochemical markers following distal forearm fracture. Twenty women (mean age 63 years) were recruited following fracture of the distal radius and ulna. Bone mineral density (BMD) of the hand and forearm were measured by dual-energy X-ray absorptiometry (DXA) and quantitative ultrasound (QUS) of the fingers was measured at 0, 6, 12, 26 and 52 weeks after fracture. Serum and urine samples were collected at 0, 3 and 7 days and at 2, 4, 6, 12, 26 and 52 weeks after fracture to measure markers of bone turnover. For bone formation we measured: bone alkaline phosphatase (iBAP), osteocalcin (Oc), procollagen type I N-terminal propeptide (PINP); and for bone resorption: tartrate-resistant acid phosphatase (TRAcP), free deoxypyridinoline (iFDpd), N-telopeptides of type I collagen (NTx). We used the nonfractured limb to calculate values for baseline BMD and amplitude-dependent speed of sound (AD-SoS). There was a decrease in BMD at the hand and in AD-SoS of the fingers after forearm fracture (p〈0.001). This bone loss was maximal for BMD by 6 weeks at 9% (p〈0.001) and remained decreased at 52 weeks. AD-SoS decreased at 12 weeks by 3% (p〈0.01) and recovered completely by 52 weeks. Bone formation markers increased between 2 and 4 weeks by 13–52% (p〈0.001), and were still elevated at 52 weeks. Bone resorption markers increased between 2 and 6 weeks by 18–35% and returned to baseline at 52 weeks (TRAcP remained elevated). We conclude that BMD decreased distal and immediately proximal to the fracture line when measured with DXA and QUS. Bone loss after distal forearm fracture did not recover by 52 weeks and most bone turnover markers did not return to baseline.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s001980050246
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Protein synthesis in skeletal muscle measured at different times during a 24 hour period (1986)
    Springer
    Bioscience reports 6 (1986), S. 209-213 
    Details
    ISSN: 1573-4935
    Keywords: protein synthesis ; muscle ; skeletal muscle
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract Six groups of 5 male rats (starting body weight 109 g) were allowed free access to a conventional rat diet. At 4 hourly intervals, starting at 10.00 h muscle protein synthesis was measured. By relating the weights of the gastrocnemius and soleus muscles to the initial body weights of the animals (i.e., at 09.30, day 1), a linear increase in muscle weight throughout the day was demonstrated. The fractional rate of muscle protein synthesis varied from 16.8% per day to 20.3% per day in gastrocnemius muscle and from 17.9% per day and 22.1% per day in the soleus. It was calculated that the maximum error incurred in estimating daily muscle protein synthesis by extrapolation of the value at any one time was 6% in gastrocnemius and 9% in soleus. It is concluded that calculations of the average rate of muscle protein degradation based on the difference between the rates of synthesis and deposition are generally valid in rats allowed free access to an adequate diet.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01115008
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  • 3
    Electronic Resource
    Electronic Resource
    Inhibition and reversal of denervation-induced atrophy by theβ-agonist growth promoter, clenbuterol (1986)
    Springer
    Bioscience reports 6 (1986), S. 811-818 
    Details
    ISSN: 1573-4935
    Keywords: muscle ; protein degradation ; clenbuterol ; denervation-induced atrophy
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract Dietary administration of the growth promoter, clenbuterol, ameliorated denervation-induced atrophy in rat soleus muscles. In acutely denervated muscles the drug inhibited the appearance of atrophy, and in chronically denervated muscles the atrophy was almost fully reversed. Responses in slow twitch oxidative fibres were particularly marked.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01117104
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  • 4
    Electronic Resource
    Electronic Resource
    Propranolol apparently separates the physical and compositional characteristics of muscle growth induced by clenbuterol (1987)
    Springer
    Bioscience reports 7 (1987), S. 51-57 
    Details
    ISSN: 1573-4935
    Keywords: muscle ; growth ; adrenergic agents ; clenbuterol ; propranolol
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The effect of propranolol on clenbuterol-induced changes in muscle fibre size and protein content were studied. Propranolol did not inhibit the ability of clenbuterol to stimulate protein accretion but reduced the increase in muscle fibre size. The compositional and physical characteristics of clenbuterol-induced muscle growth thus appeared to be separated by propranolol.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01122727
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  • 5
    Electronic Resource
    Electronic Resource
    The effect of the anabolic agent, clenbuterol, on overloaded rat skeletal muscle (1987)
    Springer
    Bioscience reports 7 (1987), S. 143-149 
    Details
    ISSN: 1573-4935
    Keywords: compensatory hypertrophy ; muscle ; clenbuterol
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The dietary administration of clenbuterol to young male rats has been shown to produce a muscle specific hypertrophic growth response. This paper demonstrates that the combined effect of drug treatment and hypertrophic stimulus induced by tenotomy produced an additive effect on muscle growth. This effect was demonstrated in terms of both muscle composition (protein and RNA) and fibre size.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01121878
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    Paper (German National Licenses)
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