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  • 1
    Online Resource
    Online Resource
    Effects of shading and micro‐spray of canopy on photosynthetic characteristics, quality and yield of drip‐irrigated grapes *
    Wiley ; 2022
    In:  Irrigation and Drainage Vol. 71, No. 1 ( 2022-02), p. 23-34
    Details
    In: Irrigation and Drainage, Wiley, Vol. 71, No. 1 ( 2022-02), p. 23-34
    Abstract: Les raisins sont sujets à une dépression de la photosynthèse (MDP) sous l'action d'une température élevée et d'une lumière forte, ce qui peut réduire l'accumulation de matière organique, entraînant une baisse du rendement et de la qualité. Dans cette étude, les raisins du Shanshan County, Xinjiang, en Chine ont été pris comme exemple pour mettre en œuvre des mesures d'ombrage et de micro‐pulvérisation. Des degrés d'ombrage de 0, 15 et 30% ont été combinés avec des débits de micro‐spray de 20, 30 et 40 L h‐1. Neuf traitements et un traitement de contrôle (le traitement de contrôle est sans ombrage et pulvérisation par brouillard) ont été conçus en combinant les conditions d'ombrage et de micropulvérisation. Le modèle de prévision photosynthétique du modèle non linéaire multivarié a été construit avec une grande précision de simulation. Le déclin de la température de l'air au sommet du couvert végétal augmentait à mesure que le degré d'ombrage et la quantité de micropulvérisation augmentaient; le MDP pourrait être éliminé dans les conditions combinées d'ombrage et de micro‐spray. Le taux d'assimilation quotidien net de CO 2 (An) augmentait de plus de 0.53 mol m‐2 s‐1. La combinaison des conditions d'ombrage et de micro‐spray a significativement augmenté la taille des fruits et favorisé la verrerie. Le traitement WP1 (15% et 40 L h‐1) a enregistré le volume annuel moyen le plus élevé par jour et le volume de fruits le plus élevé. La teneur en vitamine C (VC) du traitement par le WP1 était la plus élevée, soit 4.13 mg de kg‐1, soit 1,2 fois celle du traitement par le CK. Le rendement du traitement par le WP1 était le plus élevé, soit 19.7% de plus que celui du traitement par le CK. Ainsi, le traitement WP1 était le traitement optimal pour la promotion de l'An des feuilles de vignes et de l'amélioration de la qualité des fruits. Cette recherche pourrait fournir une référence pour améliorer la photosynthèse, la qualité des fruits et le rendement des raisins et atténuer la photosynthèse MDP des cultures.
    Type of Medium: Online Resource
    ISSN: 1531-0353 , 1531-0361
    URL: Issue
    URL: Article
    DOI: 10.1002/ird.v71.1
    DOI: 10.1002/ird.2629
    Language: English
    Publisher: Wiley
    Publication Date: 2022
    detail.hit.zdb_id: 2045607-4
    SSG: 21
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  • 2
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    Online Resource
    Intermittent short‐duration reoxygenation protects against simulated high altitude‐induced pulmonary hypertension in rats
    Wiley ; 2021
    In:  The FASEB Journal Vol. 35, No. 2 ( 2021-02)
    Details
    In: The FASEB Journal, Wiley, Vol. 35, No. 2 ( 2021-02)
    Type of Medium: Online Resource
    ISSN: 0892-6638 , 1530-6860
    URL: Issue
    URL: Article
    DOI: 10.1096/fsb2.v35.2
    DOI: 10.1096/fj.202000533RR
    Language: English
    Publisher: Wiley
    Publication Date: 2021
    detail.hit.zdb_id: 1468876-1
    SSG: 12
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  • 3
    Online Resource
    Online Resource
    Caveolae regulate vasoconstriction of conduit arteries to angiotensin II in hindlimb unweighted rats
    Wiley ; 2015
    In:  The Journal of Physiology Vol. 593, No. 20 ( 2015-10-15), p. 4561-4574
    Details
    In: The Journal of Physiology, Wiley, Vol. 593, No. 20 ( 2015-10-15), p. 4561-4574
    Abstract: Exposure to microgravity induces postflight orthostatic intolerance on re‐exposure to 1  G gravity which is second to the structural and functional remodelling of arteries. We found the maximal developed force ( E max ) of angiotensin II‐elicited vasoconstriction was decreased in abdominal aorta, unchanged in thoracic aorta and increased in carotid artery by simulated weightlessness. However, the sensitivity of the response (EC 50 ) was decreased in all of the arteries as was the desensitization of angiotensin II type I receptor (AT 1 ) upon angiotensin II stimulation. We demonstrate that caveolae on vascular smooth muscle cells play a key role in the adaptation of EC 50 and AT 1 desensitization, but not E max of the response to simulated weightlessness. This study gives insight into the mechanism underlying the arterial functional remodelling during weightlessness. Further, the findings might stimulate new ideas for research into countermeasures to postflight orthostatic intolerance upon astronauts returning to the earth. Abstract Weightlessness induces the functional remodelling of arteries, but the changes to angiotensin II (Ang II)‐elicited vasoconstriction and the underlying mechanism have never been reported. Caveolae are invaginations of the cell membrane crucial for the contraction of vascular smooth muscle cells, so we investigated the adaptation of Ang II‐elicited vasoconstriction to simulated weightlessness and the role of caveolae in it. The 4 week hindlimb unweighted (HU) rat was used to simulate the effects of weightlessness. Ang II‐elicited vasoconstriction was measured by isometric force recording. The morphology of caveolae was examined by transmission electron microscope. The binding of the angiotensin II type 1 receptor (AT 1 ) and caveolin‐1 (cav‐1) was examined by coimmunoprecipitation and Western blot. We found that the maximal developing force ( E max ) of Ang II‐elicited vasoconstriction was decreased in abdominal aorta by 30.6%, unchanged in thoracic aorta and increased in carotid artery by 17.9% after HU, while EC 50 of the response was increased in all three arteries ( P   〈  0.05). AT 1 desensitization upon activation was significantly reduced by HU in all three arteries, as was the number of caveolae ( P   〈  0.05). Furthermore, Ang II promoted the binding of AT 1 and cav‐1 significantly in control but not HU arteries. Both the number of caveolae and the binding of AT 1 and cav‐1 in HU arteries were restored by cholesterol pretreatment which also reinstated the change in EC 50 as well as the level of AT 1 desensitization. These results indicate that modified caveolae in vascular smooth muscle cells could interfere with the binding of AT 1 and cav‐1 mediating the adaptation of Ang II‐elicited vasoconstriction to HU.
    Type of Medium: Online Resource
    ISSN: 0022-3751 , 1469-7793
    URL: Issue
    URL: Article
    DOI: 10.1113/tjp.2015.593.issue-20
    DOI: 10.1113/JP270823
    RVK:
    WA 15000
    Language: English
    Publisher: Wiley
    Publication Date: 2015
    detail.hit.zdb_id: 1475290-6
    SSG: 12
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  • 4
    Online Resource
    Online Resource
    Synthesis of millimeter‐sized hydrogel beads by inverse Pickering polymerization using starch‐based nanoparticles as emulsifier
    Wiley ; 2020
    In:  Polymers for Advanced Technologies Vol. 31, No. 6 ( 2020-06), p. 1321-1329
    Details
    In: Polymers for Advanced Technologies, Wiley, Vol. 31, No. 6 ( 2020-06), p. 1321-1329
    Abstract: The preparation of millimeter‐sized poly(acrylamide‐co‐acrylic acid) hydrogel beads via inverse Pickering emulsion polymerization using starch‐based nanoparticles (SNPs) as stabilizers is reported. Amphiphilic starch is fabricated by the introduction of butyl glycidyl ether groups and palmitate groups, and the hydrophobically modified SNPs are fabricated by a nanoprecipitation process. The obtained SNPs could adsorb at oil‐water interfaces to stabilize an inverse Pickering emulsion, and the effects of oil/water volume fraction ratio and SNP concentration on emulsions are comprehensively studied. Poly(acrylamide‐co‐acrylic acid) hydrogel beads with a size of approximately 1 mm are obtained by inverse Pickering emulsion polymerization stabilized by SNPs. The morphology and structure of hydrogel beads are extensively investigated, which confirms that SNPs locate on the surface of hydrogel beads and act as emulsifiers and network structures present inside the beads. Polymerization is also detected to investigate the potential formation mechanism of hydrogel beads. The pH‐responsive property of hydrogel beads and its potential application for drug delivery are also explored.
    Type of Medium: Online Resource
    ISSN: 1042-7147 , 1099-1581
    URL: Issue
    URL: Article
    DOI: 10.1002/pat.v31.6
    DOI: 10.1002/pat.4861
    RVK:
    VA 6586
    Language: English
    Publisher: Wiley
    Publication Date: 2020
    detail.hit.zdb_id: 1481102-9
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  • 5
    Online Resource
    Online Resource
    Design of Intelligent Protective Composite Material with Stress Rate Sensitivity, Strong Interface Adhesion, and Recyclability
    Wiley ; 2023
    In:  Macromolecular Rapid Communications Vol. 44, No. 18 ( 2023-09)
    Details
    In: Macromolecular Rapid Communications, Wiley, Vol. 44, No. 18 ( 2023-09)
    Abstract: Poly(dimethyl siloxane) (PDMS) elastomers play a significant role in smart materials, actuators, and flexible electronics. However, current PDMS lacks adhesion abilities and intelligent responsive properties, which limit its further application. In this study, the polydimethylsiloxane‐ureidopyrimidinone impact hardening polymer (PDMS‐UI) composites are manufactured by a dual cross‐linking compositing tactic. PDMS, a chemically stable cross‐linked network, acts as a framework owing to its excellent mechanical strength, whereas UI, a reversible dynamic physically cross‐linked network with quadruple hydrogen bonding, endows the PDMS‐UI with excellent self‐healing ability (efficiency 〉 90%) and energy absorption (75.23%). Impressively, owing to multivalent hydrogen bonds, the PDMS‐UI exhibits superior adhesion performance: the adhesion strength on various substrates exceed 150 kPa and that on the Ferrum substrate reaches 570 kPa. These outstanding properties make the PDMS‐UI a potential candidate for application in both well‐developed fields, such as, wearable protective materials, artificial skin and soft robotics.
    Type of Medium: Online Resource
    ISSN: 1022-1336 , 1521-3927
    URL: Issue
    URL: Article
    DOI: 10.1002/marc.v44.18
    DOI: 10.1002/marc.202300216
    Language: English
    Publisher: Wiley
    Publication Date: 2023
    detail.hit.zdb_id: 1475027-2
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  • 6
    Online Resource
    Online Resource
    Heme oxygenase‐1 alleviates eosinophilic inflammation by inhibiting STAT3‐SOCS3 signaling
    Wiley ; 2020
    In:  Pediatric Pulmonology Vol. 55, No. 6 ( 2020-06), p. 1440-1447
    Details
    In: Pediatric Pulmonology, Wiley, Vol. 55, No. 6 ( 2020-06), p. 1440-1447
    Abstract: Airway inflammation of eosinophilic asthma (EA) attributes to Th2 response, leaving the role of Th17 response unknown. Signal transducer and activator of transcription 3 (STAT3) induce both suppressors of cytokine signaling 3 (SOCS3) and retinoic acid receptor‐related orphan nuclear receptor γ (RORγt) to initiate Th17 cell differentiation which is inhibited by SOCS3, a negative feedback regulator of STAT3. Heme oxygenase‐1 (HO‐1) is a stress‐responsive, cytoprotective, and immunoregulatory molecular. Two other isoforms of the enzyme includes HO‐2 and HO‐3. Because HO‐2 does not exhibit stress‐related upregulation and distributes mainly in nervous system and HO‐3 shows a low enzymatic activity, we tested a hypothesized anti‐inflammatory role for HO‐1 in EA by inhibiting STAT3‐SOCS3 signaling. Animal model was established with Ovalbumin in wild type Balb/C mice. Hemin or SNPP was intraperitoneally (IP) injected ahead of the animal model to induce or inhibit HO‐1 expression. Airway inflammation was evaluated by bronchoalveolar lavage, hematoxyline and eosin staining, enzyme‐linked immunosorbent assay, and Western blot analysis. In vivo results showed that HO‐1 induction inhibited phosphorylation of STAT3 and expression of SOCS3 and RORγt, decreased Th2 and Th17 immune responses, and alleviated airway inflammation. In vitro results revealed that HO‐1 inhibited phosphorylation of STAT3 and expression of SOCS3 in naive CD4 + T cells. These findings identify HO‐1 induction as a potential therapeutic strategy for EA treatment by reducing STAT3 phosphorylation, STAT3‐SOCS3‐mediated Th2/Th17 immune responses, and ultimate allergic airway inflammation.
    Type of Medium: Online Resource
    ISSN: 8755-6863 , 1099-0496
    URL: Issue
    URL: Article
    DOI: 10.1002/ppul.v55.6
    DOI: 10.1002/ppul.24759
    Language: English
    Publisher: Wiley
    Publication Date: 2020
    detail.hit.zdb_id: 1491904-7
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  • 7
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    Online Resource
    BK Ca compensates impaired coronary vasoreactivity through RhoA/ROCK pathway in hind‐limb unweighted rats
    Wiley ; 2019
    In:  The FASEB Journal Vol. 33, No. 12 ( 2019-12), p. 13358-13366
    Details
    In: The FASEB Journal, Wiley, Vol. 33, No. 12 ( 2019-12), p. 13358-13366
    Abstract: Previous studies have demonstrated cardiac and vascular remodeling induced by microgravity exposure. Yet, as the most important branch of vasculatures circulating the heart, the coronary artery has been seldomly studied about its adaptations under microgravity conditions. Large‐conductance Ca 2+ ‐activated potassium channel (BK Ca ) and the Ras homolog family member A (RhoA)/Rho kinase (ROCK) pathway play key roles in control of vascular tone and mediation of microgravity‐induced vascular adjustments. Therefore, we investigated the adaptation of coronary vasoreactivity to simulated microgravity and the role of BK Ca and the RhoA/ROCK pathway in it. Four‐week‐old hind‐limb unweighted (HU) rats were adopted to simulate effects of microgravity. Right coronary artery (RCA) constriction was measured by isometric force recording. The activity and expression of BK Ca and the RhoA/ROCK pathway were examined by Western blot, patch‐clamp recordings, and immunoprecipitation. We found HU significantly decreased RCA vasoconstriction to KCl, serotonin, and U‐46619, but increased protein expression and current densities of BK Ca , inhibition of which by iberiotoxin (IBTX) further decreased RCA vasoconstriction ( P 〈 0.05). Expression of RhoA and ROCK as well as active RhoA and phosphorylation of myosin light chain (MLC) at Ser 19 and MLC phosphatase target‐1 at Thr 696 were significantly increased by HU, and ROCK inhibitor Y‐27632 exerted greater suppressing effect on HU RCA vasoconstriction than that of control ( P 〈 0.05). BK Ca opener NS1619 increased HU RCA vasoconstriction, which was blocked by both RhoA and ROCK inhibitor, similar to the effect of IBTX. These results indicate that HU impairs coronary vasoconstriction but enhances BK Ca activity acting as a protective mechanism avoiding excessive decrease of coronary vasoreactivity through activation of the RhoA/ROCK pathway.—Wu, Y., Yue, Z., Wang Q., Lv, Q., Liu, H., Bai, Y. Li S. Xie M. Bao J. Ma J. Zhu X. Wang Z. BK Ca compensates impaired coronary vasoreactivity through RhoA/ROCK pathway in hind‐limb unweighted rats. FASEB J. 33, 13358–13366 (2019). www.fasebj.org
    Type of Medium: Online Resource
    ISSN: 0892-6638 , 1530-6860
    URL: Issue
    URL: Article
    DOI: 10.1096/fsb2.v33.12
    DOI: 10.1096/fj.201901273R
    Language: English
    Publisher: Wiley
    Publication Date: 2019
    detail.hit.zdb_id: 1468876-1
    SSG: 12
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  • 8
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    Free-radical solution copolymerization of the ionic liquid monomer 1-vinyl-3-ethylimidazolium bromide with acrylonitrile
    Wiley ; 2009
    In:  Journal of Applied Polymer Science Vol. 112, No. 6 ( 2009-06-15), p. 3337-3340
    Details
    In: Journal of Applied Polymer Science, Wiley, Vol. 112, No. 6 ( 2009-06-15), p. 3337-3340
    Type of Medium: Online Resource
    ISSN: 0021-8995 , 1097-4628
    URL: Issue
    URL: Article
    DOI: 10.1002/app.v112:6
    DOI: 10.1002/app.29598
    Language: English
    Publisher: Wiley
    Publication Date: 2009
    detail.hit.zdb_id: 1491105-X
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