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  • Ovid Technologies (Wolters Kluwer Health)  (31)
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  • Ovid Technologies (Wolters Kluwer Health)  (31)
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  • 1
    Online Resource
    Online Resource
    Metabolic Effects of Temocapril in Hypertensive Patients with Diabetes Mellitus Type 2
    Ovid Technologies (Wolters Kluwer Health) ; 1999
    In:  Journal of Cardiovascular Pharmacology Vol. 33, No. 4 ( 1999-04), p. 527-533
    Details
    In: Journal of Cardiovascular Pharmacology, Ovid Technologies (Wolters Kluwer Health), Vol. 33, No. 4 ( 1999-04), p. 527-533
    Type of Medium: Online Resource
    ISSN: 0160-2446
    URL: Article
    DOI: 10.1097/00005344-199904000-00003
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1999
    detail.hit.zdb_id: 2049700-3
    SSG: 15,3
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  • 2
    Online Resource
    Online Resource
    Lasting improvement of renovascular hypertension by transluminal dilatation of atherosclerotic and nonatherosclerotic renal artery stenoses. A follow-up study.
    Ovid Technologies (Wolters Kluwer Health) ; 1982
    In:  Circulation Vol. 65, No. 3 ( 1982-03), p. 611-617
    Details
    In: Circulation, Ovid Technologies (Wolters Kluwer Health), Vol. 65, No. 3 ( 1982-03), p. 611-617
    Abstract: Sixteen consecutive patients with renovascular hypertension were treated by transluminal dilatation and observed during 6 - 39 months (mean 21.8 months). Poststenotic renal artery pressure increased (p less than 0.001) and the renal arteries were patent on angiograms taken immediately after dilatation. In 13 patients, angiography was repeated 2 - 9 months later; at that time the selective renal vein renin ratio had decreased (p less than 0.001). At the end of the follow-up, blood pressure was improved or normal in 14 cases. One of the eight patients with atherosclerosis was normotensive without treatment, compared with five of six patients with fibromuscular dysplasia (p less than 0.05). The results in two cases with vasculitis are uncertain. The four patients with relapses, one after intimal catheter dissection, were treated successfully by redilatation. Thus, renovascular hypertension can be improved by transluminal dilatation in patients with atherosclerosis and in patients with fibromuscular dysplasia with lasting success and a low morbidity rate.
    Type of Medium: Online Resource
    ISSN: 0009-7322 , 1524-4539
    URL: Article
    DOI: 10.1161/01.CIR.65.3.611
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1982
    detail.hit.zdb_id: 1466401-X
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  • 3
    Online Resource
    Online Resource
    Effects of Angiotensin II-Receptor Blockade with Losartan on Insulin Sensitivity, Lipid Profile, and Endothelin in Normotensive Offspring of Hypertensive Parents
    Ovid Technologies (Wolters Kluwer Health) ; 1998
    In:  Journal of Cardiovascular Pharmacology Vol. 31, No. 4 ( 1998-04), p. 576-580
    Details
    In: Journal of Cardiovascular Pharmacology, Ovid Technologies (Wolters Kluwer Health), Vol. 31, No. 4 ( 1998-04), p. 576-580
    Type of Medium: Online Resource
    ISSN: 0160-2446
    URL: Article
    DOI: 10.1097/00005344-199804000-00016
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1998
    detail.hit.zdb_id: 2049700-3
    SSG: 15,3
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  • 4
    Online Resource
    Online Resource
    Cardiovascular, Endocrine and Renal Effects of Atrial Natriuretic Peptide in Essential Hypertension
    Ovid Technologies (Wolters Kluwer Health) ; 1986
    In:  Journal of Cardiovascular Pharmacology Vol. 8, No. 6 ( 1986-11), p. 1303-
    Details
    In: Journal of Cardiovascular Pharmacology, Ovid Technologies (Wolters Kluwer Health), Vol. 8, No. 6 ( 1986-11), p. 1303-
    Type of Medium: Online Resource
    ISSN: 0160-2446
    URL: Article
    DOI: 10.1097/00005344-198611000-00095
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1986
    detail.hit.zdb_id: 2049700-3
    SSG: 15,3
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  • 5
    Online Resource
    Online Resource
    Antihypertensive contribution of sodium depletion and the sympathetic axis during chronic angiotensin II converting enzyme inhibition
    Ovid Technologies (Wolters Kluwer Health) ; 1989
    In:  Journal of Hypertension Vol. 7, No. 11 ( 1989-11), p. 901-907
    Details
    In: Journal of Hypertension, Ovid Technologies (Wolters Kluwer Health), Vol. 7, No. 11 ( 1989-11), p. 901-907
    Type of Medium: Online Resource
    ISSN: 0263-6352
    URL: Article
    DOI: 10.1097/00004872-198911000-00009
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1989
    detail.hit.zdb_id: 2017684-3
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  • 6
    Online Resource
    Online Resource
    Pressure dependence of atrial natriuretic peptide during norepinephrine infusion in humans.
    Ovid Technologies (Wolters Kluwer Health) ; 1987
    In:  Hypertension Vol. 10, No. 3 ( 1987-09), p. 249-253
    Details
    In: Hypertension, Ovid Technologies (Wolters Kluwer Health), Vol. 10, No. 3 ( 1987-09), p. 249-253
    Abstract: The relative contribution of increased blood pressure (BP) or norepinephrine (NE), or both, to the stimulatory effect of an NE pressor infusion on circulating immunoreactive atrial natriuretic peptide (ANP) was evaluated in 10 healthy young men. They were studied during an infusion of NE, which was applied initially alone and then in combination with sodium nitroprusside. NE infusion rate was increased in four 30-minute intervals to a final dose of 200 ng/kg body weight per minute, leading to 12-fold higher plasma NE levels than were seen during control conditions. This increased mean BP (from a mean basal value of 94 +/- 3 to 119 +/- 4 [SEM] mm Hg; p less than 0.001) and plasma immunoreactive ANP (from 50 +/- 7 to 112 +/- 17 pg/ml; p less than 0.001), whereas heart rate decreased (p less than 0.001). The NE infusion was continued at the highest dose and an additional infusion of sodium nitroprusside was started to titrate mean BP in 30-minute intervals down to control values; a mean sodium nitroprusside dose of 0.95 micrograms/kg/min restored mean BP to 93 +/- 4 mm Hg (p less than 0.001), decreased plasma immunoreactive ANP to basal values (51 +/- 4 pg/ml; p less than 0.001), increased heart rate (p less than 0.001), and left plasma levels of NE largely unchanged. Plasma protein and hematocrit rose about 5 to 6% (p less than 0.001) during the NE infusion and then decreased about 3 to 4% (p less than 0.001 and p less than 0.01) when sodium nitroprusside was added.(ABSTRACT TRUNCATED AT 250 WORDS)
    Type of Medium: Online Resource
    ISSN: 0194-911X , 1524-4563
    URL: Article
    DOI: 10.1161/01.HYP.10.3.249
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1987
    detail.hit.zdb_id: 2094210-2
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  • 7
    Online Resource
    Online Resource
    Cardiovascular and renal profile of acute peripheral dopamine1-receptor agonism with fenoldopam.
    Ovid Technologies (Wolters Kluwer Health) ; 1987
    In:  Hypertension Vol. 10, No. 1 ( 1987-07), p. 43-54
    Details
    In: Hypertension, Ovid Technologies (Wolters Kluwer Health), Vol. 10, No. 1 ( 1987-07), p. 43-54
    Abstract: Whether the dopaminergic system may be involved in essential hypertension is of pathogenetic as well as therapeutic interest. Therefore, we investigated in eight hypertensive and 12 normal subjects cardiovascular, endocrine, and renal responses to fenoldopam, which has been characterized experimentally as an agonist of peripheral postsynaptic dopamine1 receptors. A single oral dose of fenoldopam, 100 mg, changed blood pressure (BP) in hypertensive subjects (from 163/103 to 147/76 mm Hg; p less than 0.01 for systolic and p less than 0.001 for diastolic BP) and normal subjects (from 121/81 to 123/65 mm Hg; p less than 0.001 for diastolic BP); percentage decreases in diastolic BP averaged -20 +/- 6 and -16 +/- 7%, respectively. Fenoldopam-induced effects on other variables were similar in the two groups. Heart rate rose (p less than 0.001) on average from 69 to 92 beats/min in hypertensive and from 64 to 84 beats/min in normal subjects. Effective renal plasma flow increased (from 552 to 765 and 634 to 937 ml/min/1.73 m2; p less than 0.01), while glomerular filtration rate tended to decrease (from 121 to 99 ml/min/1.73 m2 in the hypertensive and from 119 to 97 ml/min/1.73 m2; p less than 0.001 in the normal group). Fractional sodium clearance was elevated (from 2.8 to 5.2 and 1.7 to 3.8%; p less than 0.01), as was free water clearance (from -1.7 to 0.6 and -1.7 to 0.1 ml/min/1.73 m2; p less than 0.01). Potassium clearance was largely unchanged. Plasma renin activity increased about twofold (p less than 0.01 in normal subjects), and plasma aldosterone by 40% (NS). Plasma norepinephrine levels increased twofold to 2.5-fold (p less than 0.001), and urinary norepinephrine excretion fivefold to 10-fold (p less than 0.01). Fenoldopam-induced changes were not significantly modified by intravenous and/or oral pretreatment with the dopamine-receptor antagonist metoclopramide or the cyclooxygenase inhibitor indomethacin. These findings suggest that in humans, fenoldopam may acutely override the dopaminergic antagonism of metoclopramide given in clinical dosage and that its cardiovascular and renal effects are not prostaglandin-mediated. Although acute sympathetic stimulation may be partially antagonistic, the concomitant BP-lowering, renal vasodilating, and natriuretic actions of fenoldopam represent a desirable profile of a potential antihypertensive agent.
    Type of Medium: Online Resource
    ISSN: 0194-911X , 1524-4563
    URL: Article
    DOI: 10.1161/01.HYP.10.1.43
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1987
    detail.hit.zdb_id: 2094210-2
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  • 8
    Online Resource
    Online Resource
    Alpha 1-adrenergic blockade and cardiovascular pressor responses in essential hypertension.
    Ovid Technologies (Wolters Kluwer Health) ; 1986
    In:  Hypertension Vol. 8, No. 5 ( 1986-05), p. 407-414
    Details
    In: Hypertension, Ovid Technologies (Wolters Kluwer Health), Vol. 8, No. 5 ( 1986-05), p. 407-414
    Abstract: The effects of selective alpha 1-adrenergic blockade with terazosin on blood pressure and cardiovascular pressor responsiveness were assessed in 17 subjects with mild to moderate essential hypertension (mean age, 48 +/- 2 [SEM] years). As compared with a 2-week placebo period, 8 weeks of terazosin treatment (mean dose, 10.5 +/- 1.7 mg/day) caused a fall of supine (from 153/103 +/- 3/2 to 143/96 +/- 4/2 mm Hg; p less than 0.025) and upright (from 145/106 +/- 4/2 to 131/94 +/- 5/3 mm Hg; p less than 0.01) arterial pressure; a marked blunting of cardiovascular pressor responsiveness to norepinephrine, as judged from the pressor dose (from 73 +/- 9 to 2156 +/- 496 ng/kg/min; p less than 0.02) and from the rightward shift (p less than 0.01) of the plasma concentration-blood pressure response curve; and a slight increase in plasma norepinephrine concentration (from 37.7 +/- 3.3 to 52.2 +/- 7.8 ng/dl; p less than 0.01). Heart rate, body weight, exchangeable sodium, blood volume, and norepinephrine plasma clearance; plasma epinephrine, renin, angiotensin II, and aldosterone levels; the relationships between angiotensin II-induced increases in arterial pressure or plasma aldosterone and the concomitant increments of plasma angiotensin II; and heart rate responsiveness to isoproterenol did not change significantly after terazosin treatment. These findings suggest that the fall of arterial pressure induced by selective alpha 1-adrenergic blockade in subjects with essential hypertension is associated with, and probably explained by, inhibition of alpha 1-mediated, noradrenergic-dependent vasoconstriction. alpha 1-Adrenergic receptor antagonism did not modify body sodium concentration, the adrenomedullary component of the sympathetic nervous system, angiotensin II levels, or beta-adrenergic dependent mechanisms.
    Type of Medium: Online Resource
    ISSN: 0194-911X , 1524-4563
    URL: Article
    DOI: 10.1161/01.HYP.8.5.407
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1986
    detail.hit.zdb_id: 2094210-2
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  • 9
    Online Resource
    Online Resource
    Atrial natriuretic factor in mild to moderate chronic renal failure.
    Ovid Technologies (Wolters Kluwer Health) ; 1988
    In:  Hypertension Vol. 11, No. 5 ( 1988-05), p. 483-490
    Details
    In: Hypertension, Ovid Technologies (Wolters Kluwer Health), Vol. 11, No. 5 ( 1988-05), p. 483-490
    Abstract: The relationship between kidney function and plasma immunoreactive atrial natriuretic factor (irANF) levels as well as the effects of synthetic human ANF-(99-126) were investigated in 13 patients with mild to moderate chronic renal failure. Under basal conditions, glomerular filtration rate averaged 39 +/- 5 (SEM) ml/min/1.73 m2 and blood pressure (BP) averaged 166/107 +/- 7/2 mm Hg; 12 patients were hypertensive. Plasma irANF levels were significantly increased (98 +/- 16 vs 42 +/- 4 pg/ml in healthy control subjects; p less than 0.001) and correlated (p less than 0.05-0.005) inversely with hematocrit (r = -0.65) and positively with systolic BP (r = 0.75) or fractional sodium excretion (r = 0.75). Human ANF-(99-126) infusion for 45 minutes at 0.034 microgram/kg/min augmented (p less than 0.05-0.01) diuresis and urinary sodium, chloride, calcium, phosphate, and magnesium excretion. During the subsequent 45 minutes of human ANF-(99-126) infusion at a rate of 0.077 microgram/kg/min, diuresis and electrolyte excretion remained elevated (p less than 0.05-0.01). Glomerular filtration rate and effective renal plasma flow were not significantly modified, but filtration fraction rose progressively (p less than 0.01). Human ANF-(99-126) infusion decreased BP (p less than 0.05-0.01), produced hemoconcentration (hematocrit + 7%; p less than 0.01) without negative body fluid balance, and increased (p less than 0.01-0.001) plasma norepinephrine, insulin, and serum free fatty acids; plasma aldosterone and renin activity were unaltered during but rose after cessation of human ANF-(99-126) infusion.(ABSTRACT TRUNCATED AT 250 WORDS)
    Type of Medium: Online Resource
    ISSN: 0194-911X , 1524-4563
    URL: Article
    DOI: 10.1161/01.HYP.11.5.483
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1988
    detail.hit.zdb_id: 2094210-2
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  • 10
    Online Resource
    Online Resource
    Metabolic Neutrality of Perindopril: Focus on Insulin Sensitivity in Overweight Patients with Essential Hypertension
    Ovid Technologies (Wolters Kluwer Health) ; 1996
    In:  Journal of Cardiovascular Pharmacology Vol. 27, No. 6 ( 1996-06), p. 770-776
    Details
    In: Journal of Cardiovascular Pharmacology, Ovid Technologies (Wolters Kluwer Health), Vol. 27, No. 6 ( 1996-06), p. 770-776
    Type of Medium: Online Resource
    ISSN: 0160-2446
    URL: Article
    DOI: 10.1097/00005344-199606000-00002
    Language: English
    Publisher: Ovid Technologies (Wolters Kluwer Health)
    Publication Date: 1996
    detail.hit.zdb_id: 2049700-3
    SSG: 15,3
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