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  • 1
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    Modeling the Boron-Doping Silicon Beam by a Multilayer Model (2014)
    Hindawi
    Details
    Publication Date: 2014-02-28
    Description: The boron-doping silicon beam commonly used in microdevices exhibits a nonuniform material property along its thickness or width because of the gradient of boron concentration induced by diffusion process. The constant of rigidity, one of the most important parameters of microbeam, needs to be accurately calculated and designed in the development of high precise sensors and actuators. Current design methods, mainly depending on the analytical solutions derived under the assumption of a uniform material property or some commercial software for a varied property, are not adequate and time consuming to calculate the constant of rigidity of boron-doping silicon beam. A multilayer model is proposed in this paper to replace the continuous solid model by dividing the beam into separated layers glued together. The finite element lamination method is utilized to acquire the equivalent Young modulus and moment of inertia of cross section of multilayer model. The equivalent values are calculated from double-layer structures to multilayer ones based on the small deformation theory and the material mechanics theory. The proposed method provides an effective method to design the stiffness or frequency of microdevice and its results are validated by COMSOL simulation.
    Print ISSN: 1024-123X
    Electronic ISSN: 1563-5147
    Topics: Mathematics , Technology
    Published by Hindawi
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  • 2
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    Vascular Endothelial Injury and Apoptosis in Rats with Severe Acute Pancreatitis (2015)
    Hindawi
    Details
    Publication Date: 2015-01-23
    Description: We explored mechanisms of vascular endothelial injury that lead to systemic multiple organ failure by detecting the soluble endothelial protein C receptor (sEPCR), von Willebrand factor (vWF), serum nitric oxide (NO), and tumor necrosis factor alpha (TNF-α) and Bcl-2 mRNA and Bax mRNA expression in a severe acute pancreatitis (SAP) rat model. Compared to controls, the levels of TNF-α, vWF, and sEPCR were significantly increased in the experimental group at 12 hours and 24 hours and the NO level was significantly decreased. After 12 hours, the aortic endothelial apoptosis index and Bax mRNA expression in aortic endothelial cells had increased in the experimental group, but Bcl-2 mRNA levels had decreased. All these changes appeared at both 12 h and 24 hours. The results indicated that vascular endothelial injury and apoptosis markers were elevated in SAP. Endothelial injury and increased apoptosis in the experimental group were related to the increased expression of TNF-α.
    Print ISSN: 1687-6121
    Electronic ISSN: 1687-630X
    Topics: Medicine
    Published by Hindawi
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