In:
Circulation, Ovid Technologies (Wolters Kluwer Health), Vol. 2, No. 3 ( 1950-09), p. 392-401
Abstract:
Cardiac output and blood voluine are at control levels in unanesthetized hypertensive dogs during the depressor response to acute inflammation. Hence the fall in blood pressure is due to a decreased total peripheral resistance. The accompanying renal hyperemia indicates that decreased resistance in the renal vascular bed accounts for a significant portion of the fall in total peripheral resistance. Drug studies further suggest that acute inflammation operates to lower blood pressure in hypertensive dogs via mechanisms other than complete paralysis of autonomic vasomotor activity.
Type of Medium:
Online Resource
ISSN:
0009-7322
,
1524-4539
DOI:
10.1161/01.CIR.2.3.392
Language:
English
Publisher:
Ovid Technologies (Wolters Kluwer Health)
Publication Date:
1950
detail.hit.zdb_id:
1466401-X
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