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  • Articles  (2)
  • 2005-2009
  • 1990-1994  (2)
  • 1
    Electronic Resource
    Electronic Resource
    Marine epibiosis (1990)
    Springer
    Oecologia 82 (1990), S. 275-282 
    Details
    ISSN: 1432-1939
    Keywords: Epibiosis ; Polysyncraton lacazei ; Ascidian ; Epibiotic parameters ; Antifouling potential index
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Summary Polysyncraton lacazei is a colonial tunicate (family didemnidae) living in the NW-mediterranean rocky sublitoral. A thorough scanning of numerous colonies revealed that in spite of an apparently heavy local fouling pressure only one fouling species — a kamptozoan — is encountered with some regularity on Polysyncraton. We try to define the epibiotic situation of sessile marine organisms as composed of four epibiotic parameters: longevity or exposure time (A), epibiont load (E), colonizer pool (CP) and fouling-period (FP). Subsequently, these factors are combined to propose an “Antifouling Potential” index: AFP=(1−E/CP)×A/(FP+A). This index is intended to permit evaluating the relative antifouling defense potency to be expected in a given organism in a given epibiotic situation and to compare different cases of epibiosis and fouling.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00323545
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    The imidazoline derivative calmidazolium inhibits voltage-gated Ca2+-channels and insulin release but has no effect on the phospholipase C system in insulin producing RINm5F-cells (1994)
    Springer
    Bioscience reports 14 (1994), S. 145-158 
    Details
    ISSN: 1573-4935
    Keywords: calmidazolium ; calmodulin inhibitor ; RINm5F-cells ; Ca2+-channels ; insulin release
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The present study shows that the calmodulin antagonist calmidazolium inhibited influx of Ca2+ through voltage-gated Ca2+-channels in clonal insulin producing RINm5F-cells. The mechanism of inhibition may involve both Ca2+-calmodulin-dependent protein kinases and direct binding of calmidazolium to the Ca2+-channel. Calmidazolium did not affect uptake of Ca2+ into intracellular Ca2+-pools, inositol 1,4,5-trisphosphate (InsP3) formation or action on intracellular Ca2+-pools. The calmodulin inhibitor also did not affect glucose utilization or oxidation in RINm5F-cells, speaking against an unspecific toxic effect of the compound. KCl-and ATP-stimulated insulin release from RINm5F-cells was attenuated by calmidazolium, whereas basal hormone secretion was unaffected.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01240247
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    Paper (German National Licenses)
    Fulltext
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