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    Online Resource
    Small Bowel Dysmotilities and Small Bowel Bacterial Overgrowth
    Decker Medicine ; 2016
    In:  DeckerMed Gastroenterology, Hepatology and Endoscopy ( 2016-6-1)
    Details
    In: DeckerMed Gastroenterology, Hepatology and Endoscopy, Decker Medicine, ( 2016-6-1)
    Abstract: The main role of the small bowel is nutrient absorption; its mucosa lined with villi increases the contact surface between chyme and intestinal cells. Although the small bowel is the longest segment of the gastrointestinal (GI) tract, its transit takes between 2 and 5 hours and is the shortest of the whole gut. It is the most resilient segment of the GI tract and is thus rarely affected by motility disorders. Small bowel dysmotilities comprise a group of rare disorders affecting the function of any structure of the contractile apparatus from the muscular cells, the intrinsic neurons, or the extrinsic neurons. This review covers the epidemiology, etiology, differential diagnosis, clinical manifestations, physical examination, diagnosis, and treatment of small bowel dysmotilities and bacterial overgrowth. The figure shows GI tract innervations and related pharmacologic treatments. Tables list etiologies of small bowel dysmotilities categorized as primary or secondary causes, treatment strategies related to physiologic abnormalities, and pharmacologic treatments for GI dysmotilities. Key words: chronic intestinal pseudo-obstruction, gastrointestinal dysmotility, motility disorder, small bowel dysmotility, small intestinal bacterial overgrowth This review contains 1 highly rendered figure, 3 tables, and 51 references.
    Type of Medium: Online Resource
    URL: Journal
    URL: Article
    DOI: 10.2310/GASTRO
    DOI: 10.2310/GASTRO.5443
    Language: Unknown
    Publisher: Decker Medicine
    Publication Date: 2016
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  • 2
    Online Resource
    Online Resource
    Gastroparesis
    Decker Medicine ; 2015
    In:  DeckerMed Gastroenterology, Hepatology and Endoscopy ( 2015-8-1)
    Details
    In: DeckerMed Gastroenterology, Hepatology and Endoscopy, Decker Medicine, ( 2015-8-1)
    Abstract: The gastric phase of digestion requires a tightly coordinated neuromuscular apparatus to permit appropriate timing for each step. Dysregulations in this apparatus may be related to the Cajal cells, the intrinsic enteric nervous system, the extrinsic nervous system, the muscle cells, or any combination of structures and may lead to abnormal gastric emptying, referred to as gastroparesis. Gastroparesis is idiopathic in 35 to 49.4% of cases but may also be related to diabetes, autoimmune and inflammatory conditions, multiple sclerosis, use of certain medications, and infections, among other factors. This review describes the epidemiology, etiology, pathophysiology, clinical manifestations, diagnosis, treatment, and prognosis of gastroparesis. The figure shows symptom mechanisms related to gastric physiology in gastroparesis patients. Tables list the physiopathologic mechanisms and symptoms associated with defective gastric physiologic phenomena, the classification of gastroparesis according to affected structures, and pharmacologic treatments for gastrointestinal dysmotility. This review contains 1 highly rendered figure, 3 tables, and 56 references.
    Type of Medium: Online Resource
    URL: Journal
    URL: Article
    DOI: 10.2310/GASTRO
    DOI: 10.2310/GASTRO.5442
    Language: Unknown
    Publisher: Decker Medicine
    Publication Date: 2015
    Location Call Number Limitation Availability
    BibTip Others were also interested in ...
    Online Resource
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