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  • 1
    Electronic Resource
    Electronic Resource
    Correlative retention time peak identification method for glycated haemoglobin in high-performance liquid chromatography
    Amsterdam : Elsevier
    Journal of Chromatography A 661 (1994), S. 143-151 
    Details
    ISSN: 0021-9673
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0021-9673(93)E0886-Y
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    Paper (German National Licenses)
    Fulltext
  • 2
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    Tab. 1: Proportion of glacial quartz grains contained in the deposits
    PANGAEA
    In:  Supplement to: Ito, M; Vorndran, G (1983): Glacial Geomorphology and Snow-Lines of Younger Quaternary around the Yari-Hotaka Mountain Range, Northern Alps, Central Japan. Polarforschung, 53(1), 75-89, hdl:10013/epic.29529.d001
    Details
    Publication Date: 2023-05-12
    Description: The Yari-Hotaka Mountain Range is one of the most famous formerly-glaciated areas of Japan. Many glacial landforms remain in three neighbouring U-shaped valleys, named Yarisawa, Yokoo and Migimata. Moraines and outwash terraces can be classified into four groups according to their location and to the amount of glacial quartz grains contained in the deposits. A glaciation is proved for other parts of the Northern Japanese Alps before 100 000 years B.P., but not for the Yari-Hotaka Mountain Range, because the corresponding glacial landforms cannot be found here. The oldest known Ichinomata stage before and after 60 000 years B.P. corresponds to the Yokoo glacial which is proved wirhin the whole Japanese Alps. The three younger stages, Babadaira stage (before 30 000 years B.P.), Yarisawa stage I (about 30000 years B.P.) and Yarisawa stage II (about 15000 years B.P.), belong to the Karasawa glacial. About 10 000 years B.P. the glaciers melted away. At all times the relief-influence was especially important for Ihe mass-balances of Japanese glaciers. Wind-drifted snow from the west-exposed windward slopes to the slopes in eastern (lee) exposition, and a voluminous snow accumulation by avalanches from the high rocky walls onto the glacier surfaces beneath, caused very low situated glaciers as well as low equilibrium-lines. In most cases the snow-lines were situated 100 m or more above the equilibrium-lines. During the Ichinomata stage the snow-line reached an altitude of 2400-2450 m. It rose about 100 m to the Babadaira stage, 300 m to Yarisawa stage I and about 450 m to Yarisawa stage II. At present the snow-line is situated above the Northern Japanese Alps at over 4000 m. Therefore only perennial snow-patches exist. If the snow-line would go down by a few hundred meters, this region would be highly interesting Ifor studies on the beginning of mountain glaciation.
    Keywords: Area/locality; Counting; ELEVATION; Geological sample; GEOS; Grains, counted/analyzed; Japan; Mountain Range; Name; Sand; Scanning electron microscope (SEM); Site; Yari-Hotaka
    Type: Dataset
    Format: text/tab-separated-values, 94 data points
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    DATA PANGAEA
  • 3
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    IODP Expedition 337, Shimokita Deep Coalbed Biosphere (2015)
    PANGAEA
    In:  Supplement to: Inagaki, F; Hinrichs, Kai-Uwe; Kubo, Y; Bowles, Marshall W; Heuer, Verena B; Hong, W-L; Hoshino, Tatsuhiko; Ijiri, Akira; Imachi, H; Ito, M; Kaneko, Masanori; Lever, Mark A; Lin, Yu-Shih; Methe, B A; Morita, S; Morono, Yuki; Tanikawa, Wataru; Bihan, M; Bowden, Stephen A; Elvert, Marcus; Glombitza, Clemens; Gross, D; Harrington, G J; Hori, T; Li, K; Limmer, D; Liu, Chiung-Hui; Murayama, M; Ohkouchi, Naohiko; Ono, Shuhei; Park, Young-Soo; Phillips, S C; Prieto-Mollar, Xavier; Purkey, M; Riedinger, Natascha; Sanada, Yoshinori; Sauvage, J; Snyder, Glen T; Susilawati, R; Takano, Yoshinori; Tasumi, E; Terada, Takeshi; Tomaru, Hitoshi; Trembath-Reichert, E; Wang, D T; Yamada, Y (2015): Exploring deep microbial life in coal-bearing sediment down to ~2.5 km below the ocean floor. Science, 439 (6246), 420-424, https://doi.org/10.1126/science.aaa6882
    Details
    Publication Date: 2023-04-29
    Description: Microbial life inhabits deeply buried marine sediments, but the extent of this vast ecosystem remains poorly constrained. Here we provide evidence for the existence of microbial communities in ~40° to 60°C sediment associated with lignite coal beds at ~1.5 to 2.5 km below the seafloor in the Pacific Ocean off Japan. Microbial methanogenesis was indicated by the isotopic compositions of methane and carbon dioxide, biomarkers, cultivation data, and gas compositions. Concentrations of indigenous microbial cells below 1.5 km ranged from 〈10 to ~10**4 cells cm**-3. Peak concentrations occurred in lignite layers, where communities differed markedly from shallower subseafloor communities and instead resembled organotrophic communities in forest soils. This suggests that terrigenous sediments retain indigenous community members tens of millions of years after burial in the seabed.
    Keywords: Integrated Ocean Drilling Program / International Ocean Discovery Program; IODP
    Type: Dataset
    Format: application/zip, 2 datasets
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    DATA PANGAEA
  • 4
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    C1q/TNF-related protein-1 functions to protect against acute ischemic injury in the heart [Research Communication] (2016)
    The Federation of American Societies for Experimental Biology (FASEB)
    Details
    Publication Date: 2016-03-02
    Description: Obesity is associated with an increased risk of cardiovascular disease. C1q/TNF-related protein (CTRP)-1 is a poorly characterized adipokine that is up-regulated in association with ischemic heart disease. We investigated the role of CTRP1 in myocardial ischemia injury. CTRP1-knockout mice showed increased myocardial infarct size, cardiomyocyte apoptosis, and proinflammatory gene expression after I/R compared with wild-type (WT) mice. In contrast, systemic delivery of CTRP1 attenuated myocardial damage after I/R in WT mice. Treatment of cardiomyocytes with CTRP1 led to reduction of hypoxia–reoxygenation-induced apoptosis and lipopolysaccharide-stimulated expression of proinflammatory cytokines, which was reversed by inhibition of sphingosine-1-phosphate (S1P) signaling. Treatment of cardiomyocytes with CTRP1 also resulted in the increased production of cAMP, which was blocked by suppression of S1P signaling. The antiapoptotic and anti-inflammatory actions of CTRP1 were cancelled by inhibition of adenylyl cyclase or knockdown of adiponectin receptor 1. Furthermore, blockade of S1P signaling reversed CTRP1-mediated inhibition of myocardial infarct size, apoptosis, and inflammation after I/R in vivo . These data indicate that CTRP1 protects against myocardial ischemic injury by reducing apoptosis and inflammatory response through activation of the S1P/cAMP signaling pathways in cardiomyocytes, suggesting that CTRP1 plays a crucial role in the pathogenesis of ischemic heart disease.—Yuasa, D., Ohashi, K., Shibata, R., Mizutani, N., Kataoka, Y., Kambara, T., Uemura, Y., Matsuo, K., Kanemura, N., Hayakawa, S., Hiramatsu-Ito, M., Ito, M., Ogawa, H., Murate, T., Murohara, T., Ouchi, N. C1q/TNF-related protein-1 functions to protect against acute ischemic injury in the heart.
    Print ISSN: 0892-6638
    Electronic ISSN: 1530-6860
    Topics: Biology
    Published by The Federation of American Societies for Experimental Biology (FASEB)
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  • 5
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    Omentin attenuates atherosclerotic lesion formation in apolipoprotein E-deficient mice (2016)
    Oxford University Press
    Details
    Publication Date: 2016-03-19
    Description: Aims Obesity is associated with the development of atherosclerosis. We previously demonstrated that omentin is a circulating adipokine that is downregulated in association with atherosclerotic diseases. Here, we examined the impact of omentin on the development of atherosclerosis with gain-of-function genetic manipulations and dissected its potential mechanism. Methods and results Apolipoprotein E-deficient (apoE-KO) mice were crossed with transgenic mice expressing the human omentin gene (OMT-Tg) mice in fat tissue to generate apoE-KO/OMT-Tg mice. ApoE-KO/OMT-Tg mice exhibited a significant reduction of the atherosclerotic areas in aortic sinus, compared with apoE-KO mice despite similar lipid levels. ApoE-KO/OMT-Tg mice also displayed significant decreases in macrophage accumulation and mRNA expression of proinflammatory mediators including tumour necrosis factor-α, interleukin-6, and monocyte chemotactic protein-1 in aorta when compared with apoE-KO mice. Treatment of human monocyte-derived macrophages with a physiological concentration of human omentin protein led to reduction of lipid droplets and cholesteryl ester content. Treatment with human omentin protein also reduced lipopolysaccharide-induced expression of proinflammatory genes in human macrophages. Treatment of human macrophages with omentin promoted the phosphorylation of Akt. Inhibition of Akt signalling abolished the anti-inflammatory actions of omentin in macrophages. Conclusion These data document for the first time that omentin reduces the development of atherosclerosis by reducing inflammatory response of macrophages through the Akt-dependent mechanisms.
    Print ISSN: 0008-6363
    Electronic ISSN: 1755-3245
    Topics: Medicine
    Published by Oxford University Press
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  • 6
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    Muscle-derived follistatin-like 1 functions to reduce neointimal formation after vascular injury (2014)
    Oxford University Press
    Details
    Publication Date: 2014-06-21
    Description: Aims It is well-established that exercise diminishes cardiovascular risk, but whether humoral factors secreted by muscle confer these benefits has not been conclusively shown. We have shown that the secreted protein follistatin-like 1 (Fstl1) has beneficial actions on cardiac and endothelial function. However, the role of muscle-derived Fstl1 in proliferative vascular disease remains largely unknown. Here, we investigated whether muscle-derived Fstl1 modulates vascular remodelling in response to injury. Methods and results The targeted ablation of Fstl1 in muscle led to an increase in neointimal formation following wire-induced arterial injury compared with control mice. Conversely, muscle-specific Fstl1 transgenic (TG) mice displayed a decrease in the neointimal thickening following arterial injury. Muscle-specific Fstl1 ablation and overexpression increased and decreased, respectively, the frequency of BrdU-positive proliferating cells in injured vessels. In cultured human aortic smooth muscle cells (HASMCs), treatment with human FSTL1 protein decreased proliferation and migration induced by stimulation with PDGF-BB. Treatment with FSTL1 enhanced AMPK phosphorylation, and inhibition of AMPK abrogated the inhibitory actions of FSTL1 on HASMC responses to PDGF-BB. The injured arteries of Fstl1-TG mice exhibited an increase in AMPK phosphorylation, and administration of AMPK inhibitor reversed the anti-proliferative actions of Fstl1 on the vessel wall. Conclusion Our findings indicate that muscle-derived Fstl1 attenuates neointimal formation in response to arterial injury by suppressing SMC proliferation through an AMPK-dependent mechanism. Thus, the release of protein factors from muscle, such as Fstl1, may partly explain why the maintenance of muscle function can have a therapeutic effect on the cardiovascular system.
    Print ISSN: 0008-6363
    Electronic ISSN: 1755-3245
    Topics: Medicine
    Published by Oxford University Press
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  • 7
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    C1q/Tumor Necrosis Factor-Related Protein 9 Protects against Acute Myocardial Injury through an Adiponectin Receptor I-AMPK-Dependent Mechanism [Articles] (2015)
    The American Society for Microbiology (ASM)
    Details
    Publication Date: 2015-05-19
    Description: Obesity is a risk factor for cardiovascular disease. C1q/tumor necrosis factor-related protein 9 (CTRP9) is an adipokine that is downregulated by obesity. We investigated the role of CTRP9 in cardiac injury with loss-of-function genetic manipulations and defined the receptor-mediated signaling pathway downstream of this adipokine. CTRP9-knockout (CTRP9-KO) mice at the age of 12 weeks were indistinguishable from wild-type (WT) mice under basal conditions. CTRP9-KO mice had exacerbated contractile left ventricle dysfunction following intraperitoneal injection of lipopolysaccharide (LPS) compared to WT mice. Administration of LPS to CTRP9-KO mice also resulted in increased expression of proinflammatory cytokines and oxidative stress markers in the heart compared to WT mice. Likewise, CTRP9-KO mice showed increased myocardial infarct size and elevated expression of inflammatory mediators in ischemic heart following ischemia and reperfusion compared to WT mice. Treatment of cardiac myocytes with CTRP9 protein led to suppression of LPS-induced expression of proinflammatory genes, which was reversed by blockade of AMPK or ablation of adiponectin receptor I (AdipoR1). Systemic delivery of CTRP9 attenuated LPS-induced cardiac dysfunction in WT mice but not in muscle-specific transgenic mice expressing dominant-negative mutant form of AMPK or in AdipoR1-knockout mice. CTRP9 protects against acute cardiac damage in response to pathological stimuli by suppressing inflammatory reactions through AdipoR1/AMPK-dependent mechanisms.
    Print ISSN: 0270-7306
    Electronic ISSN: 1098-5549
    Topics: Biology , Medicine
    Published by The American Society for Microbiology (ASM)
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  • 8
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    Adipose-derived protein omentin prevents neointimal formation after arterial injury [Research Communication] (2015)
    The Federation of American Societies for Experimental Biology (FASEB)
    Details
    Publication Date: 2015-01-06
    Description: Obesity is highly linked with the development of vascular diseases. Omentin is a circulating adipokine that is downregulated in patients with cardiovascular diseases. In this study, we investigated the role of omentin in regulation of vascular remodeling in response to injury. Wild-type (WT) mice were treated intravenously with adenoviral vectors encoding human omentin (Ad-OMT) or control β -gal and subjected to arterial wire injury. Ad-OMT treatment reduced the neointimal thickening and the frequencies of bromodeoxyuridine-positive proliferating cells in injured arteries. Treatment of vascular smooth muscle cells (VSMCs) with human omentin protein at a physiologic concentration led to suppression of growth and ERK phosphorylation after stimulation with various growth factors. Omentin stimulated AMPK signaling in VSMCs, and blockade of AMPK reversed omentin-mediated inhibition of VSMC growth and ERK phosphorylation. Furthermore, fat-specific human omentin transgenic (OMT-TG) mice exhibited reduced neointimal thickening and vascular cell growth following vascular injury. AMPK activation was enhanced in injured arteries in OMT-TG mice, and administration of AMPK inhibitor reversed the reduction of neointimal hyperplasia in OMT-TG mice. These data indicate that omentin attenuates neointimal formation after arterial injury and suppresses VSMC growth through AMPK-dependent mechanisms. Thus, omentin can represent a novel target molecule for the prevention of vascular disorders.—Uemura, Y., Shibata, R., Kanemura, N., Ohashi, K., Kambara, T., Hiramatsu-Ito, M., Enomoto, T., Yuasa, D., Joki, Y., Matsuo, K., Ito, M., Hayakawa, S., Ogawa, H., Murohara, T., Ouchi, N. Adipose-derived protein omentin prevents neointimal formation after arterial injury.
    Print ISSN: 0892-6638
    Electronic ISSN: 1530-6860
    Topics: Biology
    Published by The Federation of American Societies for Experimental Biology (FASEB)
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  • 9
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    A taxonomic revision of Hymenophyllaceae (2006)
    In:  Blumea - Biodiversity, Evolution and Biogeography of Plants (0006-5196) vol.51 (2006) nr.2 p.221
    Details
    Publication Date: 2015-03-06
    Description: A new classification of Hymenophyllaceae, consisting of nine genera (Hymenophyllum, Didymoglossum, Crepidomanes, Polyphlebium, Vandenboschia, Abrodictyum, Trichomanes, Cephalomanes and Callistopteris) is proposed. Every genus, subgenus and section chiefly corresponds to the monophyletic group elucidated in molecular phylogenetic analyses based on chloroplast sequences. Brief descriptions and keys to the higher taxa are given, and their representative members are enumerated, including some new combinations.
    Keywords: filmy ferns ; Hymenophyllaceae ; Hymenophyllum ; Trichomanes
    Repository Name: National Museum of Natural History, Netherlands
    Type: Article / Letter to the editor
    Format: application/pdf
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  • 10
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    Glacial Geomorphology and Snow-Lines of Younger Quaternary around the Yari-Hotaka Mountain Range, Northern Alps, Central Japan (1983)
    Alfred Wegener Institute for Polar and Marine Research & German Society of Polar Research
    In:  EPIC3Polarforschung, Bremerhaven, Alfred Wegener Institute for Polar and Marine Research & German Society of Polar Research, 53(1), pp. 75-89, ISSN: 0032-2490
    Details
    Publication Date: 2019-07-17
    Repository Name: EPIC Alfred Wegener Institut
    Type: "Polarforschung" , peerRev
    Format: application/pdf
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