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  • 1
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2012
    In:  Archives of Clinical Neuropsychology Vol. 27, No. 6 ( 2012-09-01), p. 576-685
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 27, No. 6 ( 2012-09-01), p. 576-685
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2012
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
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  • 2
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2010
    In:  Archives of Clinical Neuropsychology Vol. 25, No. 6 ( 2010-09-01), p. 475-583
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 25, No. 6 ( 2010-09-01), p. 475-583
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2010
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
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  • 3
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2011
    In:  Archives of Clinical Neuropsychology Vol. 26, No. 6 ( 2011-09-01), p. 470-567
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 26, No. 6 ( 2011-09-01), p. 470-567
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2011
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
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  • 4
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2013
    In:  Archives of Clinical Neuropsychology Vol. 28, No. 6 ( 2013-09-01), p. 518-626
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 28, No. 6 ( 2013-09-01), p. 518-626
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2013
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
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  • 5
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2014
    In:  Archives of Clinical Neuropsychology Vol. 29, No. 6 ( 2014-09-01), p. 605-606
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 29, No. 6 ( 2014-09-01), p. 605-606
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2014
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
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  • 6
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2020
    In:  Archives of Clinical Neuropsychology Vol. 35, No. 6 ( 2020-08-28), p. 912-912
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 35, No. 6 ( 2020-08-28), p. 912-912
    Abstract: Shaken baby syndrome is a form of traumatic brain injury which occurs following physical shaking of an infant. This commonly leads to lasting neurological symptoms, such as seizures, cognitive deficits, with full symptoms often not apparent until later in life. The objective of this case study is to examine the correlation between a single episode of verified shaking as a 17-month-old child and corresponding cognitive functioning in a young adult individual. Neuroimaging confirming significant brain trauma will be included. Method The individual was shaken once as a 17-month-old and had sequalae life-long cognitive deficits. Cognitive ability was assessed with a clinical interview, neurobehavioral observations, comprehensive Neuropsychological Battery. Emotional functioning was assessed by questionnaires given to the individual’s grandmother. Results Neuropsychological testing indicated severe and pervasive cognitive deficits. The individual could not write letters or numbers and had difficulty following basic instructions, despite being cooperative with testing, due to severe attentional issues. Functionally the patient could not care for himself and needed assistance with activities of daily living. Despite his cognitive impairment, the individual had a positive attitude. Clinical interview with the patient’s family indicated a strong supportive network for the individual. Conclusion This case highlights the destructive and life-long effects of shaken baby syndrome, but also demonstrates the importance of a supportive environment for such an individual. A single instance of shaking rendered the individual unable to function independently and severely cognitively impaired. However, because of the individual’s social support, he maintains a high quality of life and is set to graduate high school at a delayed pace.
    Type of Medium: Online Resource
    ISSN: 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2020
    detail.hit.zdb_id: 2003528-7
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  • 7
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2020
    In:  Archives of Clinical Neuropsychology Vol. 35, No. 6 ( 2020-08-28), p. 839-839
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 35, No. 6 ( 2020-08-28), p. 839-839
    Abstract: Individuals exposed to lead often have persistent medical issues, changes in personality, and cognitive decline, with worsening symptoms following chronic exposure. Lead poisoning in children can have lasting effects as they age. This case study examines the correlation between cognitive ability and brain damage, with demyelination, following lead exposure in an academically well performing eight-year-old. Lead poisoning was verified by medical records and neuroimaging showing demyelination is included. Method The individual was an eight-year-old male who was exposed to lead multiple times in a home environment over the course of one year starting as a two-year-old. Cognitive ability, academic achievement, and emotional development were assessed with clinical interview of the child and his mother and comprehensive test battery including cognitive and emotional measures. Testing also included appropriate imbedded and free-standing validity measures. Results Cognitive testing showed relative weakness in certain measures of verbal and visuospatial processing speed as well as indications of deficit in sustained attention. These deficits did not reach objective impairment but suggest weakness in these areas in the context of superior overall cognitive ability and academic achievement. Observation showed hyperactivity and anxious behaviors, the individual’s mother reported elevated anxiety and unusually high somatic complaints. Conclusion This pattern of scores corresponded to what is typically seen in patients with lead poisoning, suggesting an impact of lead poisoning on the individual’s neural development, potentially causing both cognitive and psychiatric deficits. This case is unique because, despite his lead poisoning, the patient performs well academically, suggesting that the brain can compensate for this impairment given the correct environment.
    Type of Medium: Online Resource
    ISSN: 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2020
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  • 8
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2020
    In:  Archives of Clinical Neuropsychology Vol. 35, No. 6 ( 2020-08-28), p. 866-866
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 35, No. 6 ( 2020-08-28), p. 866-866
    Abstract: Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL) is a genetic disease that impacts small arteries in the brain and can result in ischemic strokes, seizures, and cognitive decline. This case study of a 61-year-old male illustrates the cognitive profile of an individual with CADASIL who has not suffered a major stroke event. Neuroimaging shows subcortical compromise in a diffuse manner. Methods The patient was assessed in an out-patient institutional practice. The individual was assessed with a clinical interview and comprehensive Neuropsychological Battery. The battery included cognitive and psychological measures with free-standing and imbedded validity measures. Results Cognitive testing demonstrated significant impairment in visuo-motor processing speed, executive planning, cognitive flexibility, verbal fluency, decision making, and visual memory, and suggests that the patients abilities show deterioration of subcortical regions, and is consistent with his CADASIL diagnosis. The patient was diagnosed with Mild Neurocognitive Disorder, due to medical condition. Conclusion Individuals with CADASIL often suffer from ischemic strokes and cognitive impairment. This study suggests that despite no major stroke event, individuals with CADASIL may still exhibit cognitive decline especially in subcortical regions. This case supports the theory that progressive cognitive decline in CADASIL patients does not begin as a result of a major stroke and can continue as a distinct cause of vascular neurocognitive disorder.
    Type of Medium: Online Resource
    ISSN: 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2020
    detail.hit.zdb_id: 2003528-7
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  • 9
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2020
    In:  Archives of Clinical Neuropsychology Vol. 35, No. 6 ( 2020-08-28), p. 851-851
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 35, No. 6 ( 2020-08-28), p. 851-851
    Abstract: Sickle-cell anemia is a leading congenital cause of stroke in childhood. Such strokes can lead to cognitive deficits, stroke in the prefrontal cortex leading to inattention, impulsivity, and poor executive control. This case study examines a 23-year-old African American male who suffered from a stroke in his prefrontal cortex at age 3. Neuroimaging confirmed stroke location, but the patient had an atypical pattern of reported cognitive deficits. Method The patient was assessed in an out-patient practice. The individual was assessed with a clinical interview and comprehensive Neuropsychological Battery. The battery included cognitive and psychological measures with free-standing and imbedded validity measures. Results Cognitive testing demonstrated significant impairment in visuospatial processing speed, visual abilities, and decision making and executive control. This pattern of impairment suggests that the patient is still suffering from his stroke, which likely damaged the connective tissue of the occipital and parietal lobes, and the prefrontal cortex. The patient was diagnosed with Mild Neurocognitive Disorder in perceptual-motor and executive functioning. Conclusions These results suggest that despite the neuroplastic abilities of the brain at early ages, the neurodevelopment of individuals who suffer from stroke in early childhood is likely to be altered and contributes to cognitive impairments in early adulthood. Long-term cognitive sequelae in the form of deficits in visual perceptual skills, suggest that frontal connections to other regions in the brain may be impacted. Therefore, this case highlights that even a localized stroke in a child can lead to broader deficits if that area of the brain interacts with other areas.
    Type of Medium: Online Resource
    ISSN: 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2020
    detail.hit.zdb_id: 2003528-7
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  • 10
    Online Resource
    Online Resource
    Oxford University Press (OUP) ; 2016
    In:  Archives of Clinical Neuropsychology Vol. 31, No. 6 ( 2016-09), p. 642.3-642
    In: Archives of Clinical Neuropsychology, Oxford University Press (OUP), Vol. 31, No. 6 ( 2016-09), p. 642.3-642
    Type of Medium: Online Resource
    ISSN: 0887-6177 , 1873-5843
    Language: English
    Publisher: Oxford University Press (OUP)
    Publication Date: 2016
    detail.hit.zdb_id: 2003528-7
    SSG: 5,2
    Location Call Number Limitation Availability
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